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E C G to continue…. Interval changes assessment. PR (PQ) interval. Normally .12 s - .20 s (that is 3 – 5 mm of horizontal distance) Shorter (e.g.) in preexcitation syndromes Longer (e.g.) in AV block of first degree Dependent of the frequency For 60 beats / s is around 0.45 s.

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E c g to continue
ECG to continue….

Interval changes assessment

Interval changes assessment

Pr pq interval
PR (PQ) interval

  • Normally .12 s - .20 s

  • (that is 3 – 5 mm of horizontal distance)

  • Shorter (e.g.) in preexcitation syndromes

  • Longer (e.g.) in AV block of first degree

  • Dependent of the frequency

  • For 60 beats / s is around 0.45 s

QT interval

Preexcitation syndromes
Preexcitation syndromes

Accessory signal pathway

Orientation of QRS complex vector depends on the direction of propagation of the signal

Sy Wolff-Parkinson-White

has “delta” wave

Sy Lown-Ganong-Levin (without “delta” wave)

Risk of supraventricular paroxysmal tachycardia

Type a type b
type A type B

Reentry tachyarrythmias
Reentry tachyarrythmias

Reentry in Wolff-Parkinson-White’s Syndrome


Ectopic atrial extrasystole



Short PR

Av block
AV block

1-st degree: Long PR inteval

2-nd degree

type one

type two

3-rd degree: No connection between atria and ventriculi

Av block of 2 nd degree
AV block of 2-nd degree

Wenkebach’s periods

(Mobitz II)

Other causes of interval changes

Short PR interval

preexcitation sy,

sympathetic act.,


AV nodal rhythms from the beginning of it

Long PRinterval

AV block1-stdegree

parasympathetic act.,



medicaments(e.g. beta blockers)

Short QT intervalDigitalis,


(hyperK – tallpointing T wave)

Long QT

hypertension, after MI


(hypoK– U wave),

Congenital (risk of sudden death)

Other causes of interval changes

Qrs left ventricular overload

Sokolow’s index: R in (V5 or V6) + S inV1

> 35mm

Attention young slim individuals (heart as a voltage source is closer to the chest leads – bigger voltage on the leads without hypertrophy)

QRS – left ventricular overload

Left heart hypertrophy


Qrs right ventricular overload
QRS – right ventricular overload

Vertical electrical axis (> 100°)

in V1: R >= 7mm or qR (volume overload)

in avR: r > 4 mm

in V6: R smaller/equal S(volume overload)

Physiological Pressure overload Physiological Volume overload

Qrs right bbb
QRS - right BBB

  • Causes: Dilatation and/or overload of right heart, MI,


  • QRS > 0,11 s

  • If complete, then R’(r’) wave is bigger then R(r ) in V1

  • Repolarization changes

Qrs left bbb
QRS – left BBB

  • Causes: IHD, hypertension,


    valvular disease,


  • QRS >0,11s (with complete block)

  • Discordant T! and discordant dinivelization of ST

Qrs q wave myocardial infarction
QRS – Q wave myocardial infarction

  • In the Q-wave MI, there is necrosis throughout the cardiac wall, while in the non-Q wave, necrosis affects the endocardial zone only.

  • Pathological Q-wave

    Appears in the first 0,04 s of QRS

    Appears in the leads where there should be no Q or overlays the normal R (r) (e.g. in V1 to V5) – absence of the R-wave

    Deeper then 2mm (6mm in III)

    Q > 0,25 R for I, II, avL, (avF)

    Q > 0,15 R forV1 to V6

Qrs q wave mi
QRS – Q wave MI

  • There is no Q-wave in the beginning, but so-called “Pardee’s”wave (elevation of ST+ negative T)

  • We imagine the (left) heart as pyramid to describe the MI location.

  • Anterior

  • Septal (right)

  • Lateral (left)

  • Inferior (down side at the apex)and it’s posterior extension (close to the base of the pyramid)

Qrs serious embolism fibrosis hydropericardium
QRS – serious embolism, fibrosis, hydropericardium