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Chapter 29 Management of Patients With Complications From Heart Disease

Chapter 29 Management of Patients With Complications From Heart Disease. Heart Failure (HF). The inability of the heart to pump sufficient blood to meet the needs of the tissues for oxygen and nutrients A syndrome characterized by fluid overload or inadequate tissue perfusion

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Chapter 29 Management of Patients With Complications From Heart Disease

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  1. Chapter 29Management of Patients With Complications From Heart Disease

  2. Heart Failure (HF) • The inability of the heart to pump sufficient blood to meet the needs of the tissues for oxygen and nutrients • A syndromecharacterized by fluid overload or inadequate tissue perfusion • The term heart failure indicates myocardial disease, in which there is a problem with the contraction of the heart (systolic failure) or filling of the heart (diastolic failure). • Some cases are reversible. • Most HF is a progressive, lifelong disorder managed with lifestyle changes and medications.

  3. Pathophysiology of Heart Failure

  4. Clinical Manifestations Right Sided Left Sided Pulmonary congestion, crackles S3 or “ventricular gallop” Dyspnea on exertion (DOE) Orthopnea Dry, nonproductive cough initially Oliguria • Viscera and peripheral congestion • Jugular venous distention (JVD) • Dependent edema • Hepatomegaly • Ascites • Weight gain

  5. Nursing Process: The Care of the Patient With Heart Failure—Assessment • Focus • Effectiveness of therapy • Patient’s self-management • S&S if increased HF • Emotional or psychosocial response • Health history • PE • Mental status; lung sounds: crackles and wheezes; heart sounds: S3; fluid status or signs of fluid overload; daily weight and I&O; assess responses to medications

  6. Nursing Process: The Care of the Patient With Heart Failure—Diagnoses • Activity intolerance related to decreased CO • Excess fluid volume related to the HF syndrome • Anxiety-related symptoms related to complexity of the therapeutic regimen • Powerlessness related to chronic illness and hospitalizations • Ineffective family therapeutic regimen management

  7. Collaborative Problems and Potential Complications • Hypotension, poor perfusion, and cardiogenic shock (see Chapter 14) • Dysrhythmias (see Chapter 26) • Thromboembolism (see Chapter 30) • Pericardial effusion and cardiac tamponade

  8. Nursing Process: The Care of the Patient With Heart Failure—Planning • Goals • Promote activity and reduce fatigue • Relieve fluid overload symptoms • Decrease anxiety or increase the patient’s ability to manage anxiety • Encourage the patient to verbalize his or her ability to make decisions and influence outcomes • Educate the patient and family about management of the therapeutic regimen

  9. Activity Intolerance • Bed rest for acute exacerbations • Encourage regular physical activity; 30–45 minutes daily • Exercise training • Pacing of activities • Wait 2 hours after eating for physical activity • Avoid activities in extreme hot, cold, or humid weather • Modify activities to conserve energy • Positioning; elevation of the HOB to facilitate breathing and rest, support of arms

  10. Fluid Volume Excess • Assessment for symptoms of fluid overload • Daily weight • I&O • Diuretic therapy; timing of meds • Fluid intake; fluid restriction • Maintenance of sodium restriction

  11. Patient Education • Medications • Diet: low-sodium diet and fluid restriction • Monitoring for signs of excess fluid, hypotension, and symptoms of disease exacerbation, including daily weight • Exercise and activity program • Stress management • Prevention of infection • Know how and when to contact health care provider • Include family in education

  12. Question What evaluation most illustrates that the patient with HF has met outcomes for the nursing diagnosis “Activity intolerance related to decreased CO?” • Exhibits decreased peripheral edema • Maintains heart rate, blood pressure, respiratory rate, and pulse oximetry within the targeted range • Avoids situations that produce stress • Performs and records daily weights

  13. Answer • Maintains heart rate, blood pressure, respiratory rate, and pulse oximetry within the targeted range Rationale: Patients with HF who exhibit stable VS shows that they have been able to adjust and plan activities to include rest and allow their bodies to adjust. A decrease in peripheral edema illustrates a reduction of fluid, avoiding situations that produce stress shows a move toward decreasing anxiety, and performing and recording daily weights shows adherence to the therapeutic regimen.

  14. Medications • Angiotensin-converting enzyme (ACE) inhibitors: vasodilation; diuresis; decreases afterload; monitor for hypotension, hyperkalemia, and altered renal function; cough • Angiotensin II receptor blockers: prescribed as an alternate to ACE inhibitors; work similarly • Hydralazine and isosorbide dinitrate: alternative to ACE inhibitors • Beta-blockers: prescribed in addition to ACE inhibitors; may be several weeks before effects seen; use with caution in patients with asthma

  15. Medications (cont’d) • Diuretics: decreases fluid volume, monitor serum electrolytes • Digitalis: improves contractility, monitor for digitalis toxicity especially if patient is hypokalemic • IV medications: indicated for hospitalized patients admitted for acute decompensated HF • Milrinone: decreases preload and afterload; causes hypotension and increased risk of dysrhythmias • Dobutamine: used for patients with left ventricular dysfunction; increases cardiac contractility and renal perfusion

  16. Gerontologic Considerations • May present with atypical signs and symptoms such as fatigue, weakness, and somnolence • Decreased renal function can make older patients resistant to diuretics and more sensitive to changes in volume • Administration of diuretics to older men requires nursing surveillance for bladder distention caused by urethral obstruction from an enlarged prostate gland

  17. Question Which classification of medications play a pivotal role in the management of HF caused by systolic dysfunction? • ACE inhibitors • Beta-blockers • Diuretics • Digitalis

  18. Answer • ACE inhibitors ACE inhibitors play a pivotal role in the management of HF caused by systolic dysfunction. Beta-blockers have been found to reduce mortality and morbidity in patients with NYHA class II or III HF by reducing the adverse effects from the constant stimulation of the sympathetic nervous system. Diuretics are prescribed to reduce excess extracellular fluid by increasing the rate of urine produced in patients with signs and symptoms of fluid overload. Digitalis increases the force of myocardial contraction and slows conduction through the atrioventricular node.

  19. Pulmonary Edema • Acute event in which the LV cannot handle an overload of blood volume. Pressure increases in the pulmonary vasculature, causing fluid movement out of the pulmonary capillaries and into the interstitial space of the lungs and alveoli. • Results in hypoxemia • Clinical manifestations: restlessness, anxiety, dyspnea, cool and clammy skin, cyanosis, weak and rapid pulse, cough, lung congestion (moist, noisy respirations), increased sputum production (sputum may be frothy and blood tinged), decreased level of consciousness

  20. Management of Pulmonary Edema • Prevent • Early recognition: monitor lung sounds and for signs of decreased activity tolerance and increased fluid retention • Place patient upright and dangle legs • Minimize exertion and stress • Oxygen • Medications • Diuretics (furosemide), vasodilators (nitroglycerin)

  21. Cardiogenic Shock • A life-threatening condition with a high mortality rate • Decreased CO leads to inadequate tissue perfusion and initiation of shock syndrome. • Clinical manifestations: symptoms of HF, shock state, and hypoxia

  22. Management of Cardiogenic Shock • Correct underlying problem • Treated in an ICU • Assess cardiac rhythm, monitor hemodynamic parameters, monitor fluid status, and adjust medications and therapies based on the assessment data • Medications • Diuretics, positive inotropic agents and vasopressors • Circulatory assist devices • Intra-aortic balloon pump (IABP)

  23. Thromboembolism • Decreased mobility and decreased circulation increase the risk for thromboembolism in patient with cardiac disorders, including those with HF. • Pulmonary embolism: blood clot from the legs moves to obstruct the pulmonary vessels. • The most common thromboembolic problem with HF • Prevention • Treatment • Anticoagulant therapy

  24. Pulmonary Emboli

  25. Pericardial Effusion and Cardiac Tamponade • Pericardial effusion is the accumulation of fluid in the pericardial sac. • Cardiac tamponade is the restriction of heart function because of this fluid, resulting in decreased venous return and decreased CO. • Clinical manifestations: ill-defined chest pain or fullness, pulsus paradoxus, engorged neck veins, labile or low BP, shortness of breath • Cardinal signs of cardiac tamponade: falling systolic BP, narrowing pulse pressure, rising venous pressure, distant heart sounds

  26. Assessment Findings in Cardiac Tamponade

  27. Medical Management • Pericardiocentesis • Pericardiotomy

  28. Sudden Cardiac Death or Cardiac Arrest • Emergency management: cardiopulmonary resuscitation • A: airway • B: breathing • C: circulation • D: defibrillation for VT and VF

  29. Question What is the most reliable sign of cardiac arrest in an adult and child? • Decrease in blood pressure • Absence of brachial pulse • Absence of breathing • Absence of carotid pulse

  30. Answer • Absence of carotid pulse Rationale: The most reliable sign of cardiac arrest is the absence of a pulse. In an adult or child, the carotid pulse is assessed. In an infant, the brachial pulse is assessed.

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