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Age Related Macular Degeneration Treatment In Ghatkopar, Mumbai

Human eye has various important parts like Cornea, Pupil, Iris, Lens and Retina. The macula is located in the center of the retina, the light-sensitive tissue at the back of the eye. The retina instantly converts light, or an image, into electrical impulses.

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Age Related Macular Degeneration Treatment In Ghatkopar, Mumbai

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  1. Age Related Macular Degeneration Treatment In Ghatkopar, Mumbai: • Humaneyehasvariousimportantparts likeCornea,Pupil,Iris,LensandRetina.The maculais located in the center of the retina, the light-sensitive tissue at the back of the eye. The retina instantly convertslight,oran image,intoelectricalimpulses.Theretina thensendstheseimpulses,ornerve signals,tothebrain.Whenthecellsofthemaculadeteriorate,imagesarenotreceivedcorrectly.In earlystages,maculardegeneration doesnotaffectvision.Later, ifthediseaseprogresses,people experiencewavyorblurredvision,and,iftheconditioncontinuestoworsen,centralvisionmay be completelylost.Peoplewithveryadvancedmaculardegenerationareconsideredlegally • blind.MacularDegenerationistheleadingcause ofvisionloss,more thancataractsandglaucomacombined. • Maculardegenerationisclassified as: • DryAgerelated MacularDegeneration • WetAgerelatedMacularDegeneration. • Pathophysiology • Thedryform ismorecommonthanthewetform,withabout85to90 percentofAMD patientsdiagnosedwith dryAMD.ThelesscommonwetAMDusuallyleadstomoreserious visionloss. • DryAMDcauseschangesoftheretinalpigment epithelium,typicallyvisible asdarkpinpoint areas.Theretinalpigmentepitheliumplaysacriticalroleinkeepingtheconesandrods

  2. healthyandfunctioningwell.Accumulation ofwasteproductsfrom therodsandconescan resultindrusen,whichappearasyellowspots. Areasofchorioretinalatrophy(referredtoas geographicatrophy)occur inmoreadvancedcasesofdryAMD.Thereisno elevated macular scar (disciform scar),edema,hemorrhage,or exudation. • DryAMD hasthree stages,allofwhich mayoccurinone orboth eyes: • EarlyAMD-People withearlyAMDhave eitherseveralsmalldrusenorafewmedium-sized drusen.Atthisstage,thereare nosymptomsandnovisionloss. • IntermediateAMD- Peoplewithintermediate AMDhaveeithermanymedium-sizeddrusen • or one or morelarge drusen.Somepeople see ablurred spotinthecenteroftheir vision. Morelightmaybe neededfor readingandother tasks. • Advanced AMD-Inadditiontodrusen,peoplewith advanceddryAMD haveabreakdown • oflight-sensitive cellsandsupporting tissuein the centralretinalarea.Thisbreakdowncan causea blurredspotin thecenterofyour vision.Over time,the blurredspotmaygetbigger anddarker,takingmoreofyourcentralvision. Youmayhave difficultyreading or recognizing facesuntiltheyareveryclosetoyou. • WetAMDoccurswhen newabnormalblood vesselsdevelopundertheretina in a process • called choroidalneovascularization(abnormalnewvesselformation).Localizedmacular edemaor hemorrhagemayelevate anareaofthemaculaorcausealocalizedretinal pigment epithelialdetachment.Eventually,untreatedneovascularizationcauses adisciform scarunder themacula. • Symptoms • Drymaculardegenerationsymptoms usuallydevelopgradually andwithoutpain. Theymayinclude: • Visualdistortions,such asstraightlinesseemingbent • Reducedcentralvision inone or botheyes • Theneedforbrighterlightwhenreadingordoingclosework • Increased difficultyadaptingtolowlightlevels,suchaswhen entering adimlylitrestaurant • Increasedblurrinessofprintedwords • Decreasedintensityorbrightnessofcolors • Difficultyrecognizingfaces Whatcausesmaculardegeneration?

  3. Thoughmaculardegenerationisassociatedwithaging,there isgeneticcomponenttothe disease.Astrong association between development ofAMD and presence ofavariant ofa geneknown ascomplementfactor H(CFH)isobserved.Thisgenedeficiencyisassociated withalmosthalfofallpotentiallyblindingcasesofmaculardegeneration. • Other investigatorshavefoundthatvariantsofanother gene,complementfactorB,maybe involvedindevelopmentofAMD. • Specificvariantsofone orboth ofthese genes,which playaroleinthe body'simmune responses,have beenfoundin 74percent ofAMDpatientswhowerestudied.Other complementfactorsalsomaybe associatedwithanincreasedriskofmaculardegeneration. • Oxygen-deprivedcellsin theretina produce atypeofprotein calledvascularendothelial growthfactor (VEGF),which triggersthe growth ofnewblood vesselsintheretina. • ThenormalfunctionofVEGFistocreatenewbloodvesselsduring embryonicdevelopment, after aninjuryortobypassblocked bloodvessels.ButtoomuchVEGFinthe eye causesthe developmentofunwanted blood vesselsin theretinathateasilybreakopen and bleed, damagingthemaculaandsurroundingretina. • RiskFactors • ThebiggestriskfactorforMacularDegeneration isage.Yourriskincreasesasyouage, andthediseaseismostlikelytooccurinthose 55andolder. • Otherriskfactorsinclude: • Genetics–PeoplewithafamilyhistoryofAMDareatahigher risk. • Race–Caucasiansaremorelikelyto developthe disease thanAfrican-Americansor Hispanics/Latinos. • Smoking –SmokingdoublestheriskofAMD. • Diagnosis • AMDisdetected duringa comprehensive eyeexamthatincludes: • Visualacuity test-Thiseyecharttestmeasureshow wellyousee atvariousdistances. • Dilated eyeexam- Dropsareplaced inyoureyestowidenthe pupils.Youreyecare • professionalusesa specialmagnifyinglenstoexamineyourretina and opticnerveforsigns ofAMDand othereye problems.Afterthe exam,your close-upvisionmayremain blurredfor severalhours.

  4. Tonometry -Aninstrumentmeasuresthepressureinsidetheeye.Numbing dropsmaybe appliedtoyour eyefor thistest. • Bothforms of age- relatedmaculardegeneration(AMD) arediagnosed byfunduscopic examination.Visualchangescanoftenbedetectedwith anAmslergrid. • Colorphotographyandfluoresceinangiography aredonewhenfindingssuggestwetAMD. Angiographyshowsandcharacterizessubretinalchoroidalneovascularmembranesand can delineateareasofgeographicatrophy.Opticalcoherence tomography(OCT) aidsin identifyingintraretinalandsubretinalfluidand can help assessresponsetotreatment. • What Treatments Are Available for Macular Degeneration? • There’snocureformaculardegeneration.Treatmentmayslowitdown or keepyoufrom losingtoomuch ofyourvision.Youroptionsmightinclude: • Lifestylechanges-likedieting,exercise, avoidingsmoking, andprotectingyoureyesfrom ultravioletlight. • Anti-angiogenesisdrugs-Thesemedications –aflibercept(Eylea),bevacizumab (Avastin),pegaptanib(Macugen),andranibizumab(Lucentis)--blockthe creation ofblood vessels and leaking from the vessels in your eye that cause wet macular degeneration. Manypeoplewho’ve takenthesedrugsgotbackvisionthatwaslost.Youmightneedto havethistreatmentmultipletimes. • Lasertherapy - High-energylaserlightcandestroyabnormalbloodvesselsgrowinginyour eye. • Photodynamiclasertherapy-Yourdoctor injectsalight-sensitivedrugverteporfin • (Visudyne)intoyourbloodstream, andit’sabsorbedbythe abnormalbloodvessels.Your doctor thenshinesalaserintoyoureyeto triggerthemedicationtodamagethoseblood vessels. • Lowvisionaids- Thesearethe devicesthathavespeciallensesorelectronicsystemsto createlargerimagesofnearbythings.Theyhelp peoplewho havevisionlossfrom macular degeneration makethemostoftheir remainingvision. • Submacularsurgery- Thisremovesabnormalbloodvesselsor blood.

  5. Retinaltranslocation- Aprocedureto destroyabnormalbloodvesselsunderthe centerof your macula,whereyourdoctor can’tuse alaserbeam safely.Inthisprocedure,yourdoctor rotatesthe centerofyourmacula awayfromtheabnormalbloodvesselstoahealthyareaof your retina.Thiskeepsyoufromhavingscartissueandmore damagetoyourretina.Then, yourdoctorusesalaser to treattheabnormalbloodvessels. • Important Reminder:Thisinformationisonlyintended to provide guidance,nota definitive medical advice. Please consult eye doctorabout your specific condition. Only a trained, experienced board certifiedeyedoctorcan determinean accuratediagnosisand proper treatment. ToscheduleanappointmentwithourexpertsforAgeRelatedMacularDegeneration Managementpleasecall usat+91 8451045935,+91-8451045934orvisitourclinicat Address.

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