APOPTOSIS
This study by Bolhàr-Nordenkampf and Meister from the University of Vienna explores the impacts of ozone (O3) on photosynthetic plant cells. It examines how ozone injury leads to apoptotic processes due to reactive oxygen species (ROS) overload. The research highlights the interplay between stomatal function, drought stress, and the oxidative burst that compromises plant health. Key findings reveal that stress factors inhibit CO2 uptake, affecting the Calvin Cycle and overall plant viability. This work contributes to a deeper understanding of plant responses to environmental stressors.
APOPTOSIS
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Presentation Transcript
APOPTOSIS University of Vienna Faculty of Life Science Bolhàr-Nordenkampf & Meister Society for the Advancement of Plant sciences Follow the clicks and >! Fin 9.01.2008
Ozone injury O3 *click! Uptake and Action of Ozone in photosynthetic active plant cells as model for a general Stress response. uptake “dry deposition” *click! Apoptosis is the result of an overload by reactive Oxygen species Stomata pore drought stress Apoplast Oxidative Burst Intercellular spaces *click! Cytoplasm internal free surface area Chloroplast *click! Glutathione, Ascorbate Halliwell-Cycle> Cell wall Xanthophyll-Cycle Protonation, Zeaxanthin> *click! *click! Carbonic anhydratase Tunnel proteins Assimilatory power CO2 * *click! * protection> overload *click! water from xylem Plasmalemma Sucrose Photosynthesis *click! Tocopherol ß-Carotin Zeaxanthin> Electron- and Proton transport Phloem loading *click! e.g. Starch-Biosynthesis in tubers, grain filling *click! In the end all stress factors inhibit CO2 uptake. Than the Calvin Cycle does not need anymore assimilatory power. In the light a feed back reaction creates reactive oxygen species (ROSINT) at PSI and PSII. If ROSINT overload the stress compensation capacity, the cell will get destroyed. H.R. Bolhàr-Nordenkampf M.H. Meister Society for the Advancement of Plant sciences University of Vienna, Faculty of Life Science
