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Case Study IV

Case Study IV. Joni Williamson Sean Hester. Examination. Mrs. Jenkins 29 y/o female Lives with her husband and 1 small child Stay at home mom MOI: unknown PT needed due to limited mobility PMH: ED visit 2 days ago, sent home on Vicodin, recently recovered from flu Right hand dominant.

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Case Study IV

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  1. Case Study IV Joni Williamson Sean Hester

  2. Examination • Mrs. Jenkins • 29 y/o female • Lives with her husband and 1 small child • Stay at home mom • MOI: unknown • PT needed due to limited mobility • PMH: ED visit 2 days ago, sent home on Vicodin, recently recovered from flu • Right hand dominant

  3. Examination Physical Examination: • A&O x 3 • Vitals elevated due to obvious pain • Peripheral pulses intact • Appears well-nourished and stated age • Altered facial expressions

  4. Examination Neurologic Examination: • Cranial nerves intact • Short term memory deficits found

  5. Neurological Exam Motor Systems: • R UE – STR 2/5 with spasticity • L UE – STR 4/5 with normal tone • R LE – STR 2-/5 with spasticity • L LE – STR 4/5 with normal tone • DTR: R – hyperreflexia L - WNL

  6. Neurological Exam Sensation: • Light touch test: • Pain elicited with dull (non-noxious) stimulus • Further sensory is contraindicated due to hypersensitivity of pain.

  7. Pathology • THALAMIC SYNDROME- from a CVA to the posterior cerebral artery. • Thalamic pain due to spinothalamic, VPL/VPM damage • Contralateral hemiparesis due to interuption of the corticospinal tract • Emotional/behavioral issues- due to motor involvement in the face • Choreaathetoid movements

  8. Evaluation: Disablement Model • Pathology- Thalamic Syndrome • Impairment- Decrease strength of R UE and R LE • Functional Limitation- pt is unable to ambulate • Disablity-pt can’t ambulate at home to take care of her child

  9. Prognosis • The overall prognosis for this patient is fair. • The strength in the UE and LE can be increased, but the ultimate success depends on the ability to control the pain that will be everlasting.

  10. Intervention • Pain control • Gate training • Strength training • Stretching • ROM • Pt and caregiver education • Aquatic Therapy

  11. Interventions • There is no surgical treatment for this syndrome. • A multidisciplinary approach will be needed to treat the patient. • Pain medications (prescribed and NSAIDS) have been used to manage the pain as best as possible. • In some cases medicinal marijuana is prescribed to help manage pain • Study conducted (2007) using vestibular caloric stimulation (COWS) to treat thalamic pain syndrome on two patients. • Both participants showed decrease pain immediately and long-term (4-7 weeks) after the treatment.

  12. Goals • LTG: Pt to increase strength to 4/5 in R UE and LE in 8 wks to be able to lift and care for her small child. • STG: Pt to demonstrate 3/5 strength in R UE and LE in 4 wks. • LTG: Pt to ambulate 400 ft on an even surface with a cane in 8 wks in order to independently walk from the bedroom to the kitchen and back. • STG: Pt to ambulate 100 ft on an even surface with a cane in 2 wks.

  13. APTA Practice Pattern • 5E: Impaired Motor Function and Sensory Integrity Associated With Progressive Disorders of the Central Nervous System

  14. PT Implications • Impaired motor function • Gait training • Strengthening • Stretching • ROM • Impaired sensory integrity • Pain control • Progressive loss of function • Pt and caregiver education

  15. References • Ramachandran V S, McGreoch P D, Williams L, Arcilla G. “Rapid Relief of Thalamic Pain Syndrome Induced by Vestibular Caloric Stimulation.” Neurocase 2007;13:185-188. • Schott G. From thalamic syndrome to central poststroke pain. Journal of Neurology, Neurosurgery, and Psychiatry 1995;61:560-564.

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