'Channel blockers' presentation slideshows

CLINICAL PHARMACOLOGY OF ANTIARRHYTHMIC MEDICATIONS

CLINICAL PHARMACOLOGY OF ANTIARRHYTHMIC MEDICATIONS. Vincent F. Mauro, PharmD, FCCP. Professor of Clinical Pharmacy and Adjunct Professor of Medicine The University of Toledo. GOALS. To have a better understanding of:

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CLASSIFICATION OF ANTI ARRHYTHMIC DRUGS

CLASSIFICATION OF ANTI ARRHYTHMIC DRUGS. www.anaesthesia.co.in anaesthesia.co.in@gmail.com. DENOSUMAB. - 31. - 29. Action potential. Phase 0 Na current Phase 1 Transient outward current Phase 2,3 Ca current vs k current Phase 4 Pacemaker current. Na channel blockers.

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Antidysrhythmic and Antihypertensive Agents

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Tachydysrrhythmias

Tachydysrrhythmias. Lisa Campfens MD, FRCPC, FACEP. Generation of Dysrrhythmias. Two fundamental causes Disturbances of automaticity Disturbances of conduction AV block Reentry. Presentation. Multiple symptoms: Fatigue Chest pain Dyspnea Dizziness Presyncope Palpitations

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??? ???? ?????? ??????

??? ???? ?????? ??????. Antiarrythmic Drugs ???? ?? ???? ??????? ??? ????? ??????? ????? ???? ???? ???? ?? ??????? ?????? ??? ???????... ?? ??????? ??? ??? ????????? ?????? ??? ??????? ???? ?? ?? ???? ???? ??? ????? ????? ... ?? ????? ????? 426 ???? ????? ????????? ...

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Thoracolumbar Fractures

Thoracolumbar Fractures. Patient Evaluation and Management. Outline. Epidemiology Clinical evaluation ATLS Neuro exam Neurogenic / spinal shock Classification of spinal cord injury Grading system Complete VS incomplete Incomplete cord syndromes Pharmacological treatment. Outline.

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Antiarrhythmic Drugs ???????

Antiarrhythmic Drugs ???????. Shi-Hong Zhang ( ??? ) Dept. of Pharmacology shzhang713@zju.edu.cn. Types of arrhythmia. Supraventricular arrhythmias: Atrial fibrillation, flutter Premature atrial contraction Paroxysmal supraventricular tachycardia (originating from atria or AV node).

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Prehospital Pharmacology

Prehospital Pharmacology. 6. 5. Prehospital medicating. Prescribed by GP Standing Orders Protocols Transfer orders (should be written) EMS administered. Ace Inhibitors.

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A CASE STUDY IN ION CHANNEL DRUG DISCOVERY

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Angina pectoris

Angina pectoris. Sudden, severe, pressing chest pain and radiating to the neck, jaw, back, and arms. The episodes are transient, stay between 15 sec to 15 min.

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Dr Abdelaziz Elamin, MD, PhD, FRCPCH College of Medicine Sultan Qaboos University

TUMORAL CALCINOSIS. Dr Abdelaziz Elamin, MD, PhD, FRCPCH College of Medicine Sultan Qaboos University. BACKGROUND. Virchow in 1855 used the term Calcinosis Cutis to describe deposition of calcium in the skin & S/C tissues. It is classified into 4 major types according to etiology:

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Prescription Drugs

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ANTIHYPERTENSIVES

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Cardiac Arrhythmias

Cardiac Arrhythmias. Types of cardiac arrhythmias : Bradyarrhythmias Tachyarrhythmias Bradyarrhythmias: treat with atropine, pacing Tachyarrhythmias can occur due to: Enhanced automaticity Afterdepolarization and triggered activity Re-entry. Tachyarrhythmias : Enhanced automaticity:

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Cardiac Arrhythmias

Cardiac Arrhythmias. Types of cardiac arrhythmias : Bradyarrhythmias Tachyarrhythmias Bradyarrhythmias: treat with atropine, pacing Tachyarrhythmias can occur due to: Enhanced automaticity Afterdepolarization and triggered activity Re-entry. Tachyarrhythmias : Enhanced automaticity:

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Na + channel blocker : Na + channel block depends on: HR Membrane potential

Na + channel blocker : Na + channel block depends on: HR Membrane potential Drug specific physiochemical characteristic- ? recovery Blockade of Na + channels results in: Threshold for excitability is increased (more current) Increase in pacing and defibrillation threshold

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Cardiac Action Potentials and Anti-Arrhythmic Drugs

Cardiac Action Potentials and Anti-Arrhythmic Drugs. Ca 2+ in (long). +10. K + out. Na + in (I f ) Ca2+ in (transient). -60. 4 K + (inwardly rectifying). 0 Na + FAST. 1 K + out. 2 Ca 2+ in (L-type) K + out (slow delayed). 3 K + out (slow & rapid delayed). 4

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Anti-Anginal Agents

Anti-Anginal Agents. Angina Disease, symptoms Types of Anti-anginal Agents Nitrovasodilators, calcium channel blockers and b -blockers Nitrovasodilators Structure, physical properties, biochemical mechanism of action, metabolism and pharmacokinetics Calcium channel blockers

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