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PATHOPHYSIOLOGY

PATHOPHYSIOLOGY. PRECIPITATING FACTORS. PREDISPOSING FACTORS. -AGE <20 AND 40>. -ABNORMALITY IN THE UTERUS. LOW FOLIC ACID INATAKE. -DEFECTS IN THE EGG. LOW CALORIE INTAKE. -ASIAN HERITAGE. LOW CHO INTAKE. Egg cell fails to change composition. EGGS DONOT HAVE CHROMOSOMAL PROPERTIES.

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PATHOPHYSIOLOGY

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  1. PATHOPHYSIOLOGY PRECIPITATING FACTORS PREDISPOSING FACTORS -AGE <20 AND 40> -ABNORMALITY IN THE UTERUS LOW FOLIC ACID INATAKE -DEFECTS IN THE EGG LOW CALORIE INTAKE -ASIAN HERITAGE LOW CHO INTAKE Egg cell fails to change composition EGGS DONOT HAVE CHROMOSOMAL PROPERTIES EMPTY EGG FERTILIZED BY A ½ SPERMS EGG FERTILIZED BY 2 SPERMS (+) pregnancy test FERTILIZATION OF AB ZYGOTE

  2. Trophoblastic PROLIFIRATION ABDOMINAL PAIN FASTER UTERINE GROWTH MULTIPLE THECALUTEUM INC. HCG INC. PROGESTERONE CHORIONIC TYROTROPINE DECREASE ESTROGEN ENLARGEMENT OF OVARIE CYST HYPEREMESIS AMENORRHEA CONTRACTION HYPERTHYROIDISM INC. BMR SEPERATION OF VESICLES FROM UTERINE WALL WEIGHT LOSS INC. HR/ DIAPORESIS VAGINAL BLEEDING CAN’T TOLERATE HEAT HYPERTHYROIDISM PRE-EC-DIZZINESS -HEADACHE PALLOR

  3. (+) fetal tone All vesicular chorionic villi formed (-) Fetal tone Some chorionic villi formed Profuse bleeding -BP Abnormal high HcG Incomplete implantation Some retained trophoblast becomes malignant Spontaneous abortion hyperthyroidism complications Fatigue Weight loss Muscle weakness Increase hearth rate Diaphoresis Anxiety Thyroid enlargement Choriocarcinioma Invasive mole Persistent gtd

  4. -DEFECTS IN THE EGG • PREDISPOSING FACTORS SWELLING PROTEINURIA HEADACHE CHANGES OF REFLEXES IMPLANTATION Trophoblastic maturation

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