'Acute inflammation' presentation slideshows

Otitis Media

Otitis Media. Mary Bennett, Amanda Buisman & Roline Campbell. Pertinent Anatomy. Ossicles (malleus, incus, stapes). OR Auricle. External Ear Canal. OR Tympanic Membrane. Pertinent Anatomy. (Cone of light). Physiology of the Ear. External Ear

2.38k views • 86 slides

Diseases of the conjunctiva Ayesha s Abdullah 21.09.2013

Diseases of the conjunctiva Ayesha s Abdullah 21.09.2013. LEARNING OUTCOMES . By the end of this lecture the students would be able to; Classify diseases of the conjunctiva Identify the common symptoms and signs of conjunctival diseases Classify diseases of the conjunctiva

1.33k views • 57 slides

INFLAMMATION AND REPAIR Lecture 3 Chemical Mediators in Inflammation and

Foundation block: pathology. INFLAMMATION AND REPAIR Lecture 3 Chemical Mediators in Inflammation and Patterns of Acute Inflammation. Dr. Maha Arafah Associate Professor Department of Pathology King Saud University Email: marafah@hotmail.com. Oct, 2015.

1.15k views • 52 slides

FOCAL ACTIVE COLITIS PATTERN

FOCAL ACTIVE COLITIS PATTERN. Focal active colitis (FAC) Is a histologic pattern characterized by single foci of neutrophilic crypt injury without features of chronic injury ( chronicity) The pattern encompasses a spectrum of histologic changes, ranging from

1.2k views • 39 slides

L ecturer: Dr. Maha Arafah

INFLAMMATION AND REPAIR Lecture 1 Definition of inflammation; acute inflammation Vascular Events in Inflammation. L ecturer: Dr. Maha Arafah. (Foundation Block, pathology 14/10/2014. Learning Objectives:. Define inflammation. Recognize the cardinal signs of inflammation.

1.14k views • 38 slides

Mechanisms of Disease

Mechanisms of Disease . Cell Injury, acute and chronic inflammation. Learning objectives from handbook. Question based upon learning objectives. The basics. Discuss and list the causes of cell injury and death. Hypoxia Physical agents Chemical agents Micro-organisms Immune mechanisms.

587 views • 34 slides

Testicular Pathology

Testicular Pathology. Emad Raddaoui , MD, FCAP, FASC Associate Professor & Consultant. Normal Anatomy. Testicular diseases. Epididymitis And ORCHITIS : Inflammatory conditions are generally more common in the epididymis than in the testis.

643 views • 34 slides

Normal pancreas

Normal pancreas. Pathology of pancreas. P ancreatitis . Inflammation of the pancreas May be diffuse or focal Acute or chronic

541 views • 29 slides

Diagnosis of pulmonary aspergillosis (ignoring allergy)

Diagnosis of pulmonary aspergillosis (ignoring allergy). David W. Denning Wythenshawe Hospital University of Manchester. Normal. Massive. Hyphal load in tissue. Immune function. Conceptual framework. Chronic inflammation and fibrosis. Vascular invasion, necrosis, dissemination.

484 views • 27 slides

Lab cases

Lab cases. Dr : MARAM YOUSEF. 1. Atrophy . Atrophy. Cardiac hypertrophy. Prostatic hyperplasia. Hyperplastic prostate. Metaplasia. Intracellular accumulations: fatty change.

403 views • 23 slides

Acute appendicitis

1.21k views • 22 slides

INTRODUCTION TO THE IMMUNE SYSTEM

INTRODUCTION TO THE IMMUNE SYSTEM. Janet Liversidge Department of Ophthalmology Medical School j.liversidge@abdn.ac.uk. Innate and adaptive immunity. The course of a typical antibody response: The adaptive response takes 7-14 days to develop and mount a specific, protective immune response.

884 views • 19 slides

Week 2- Acne

Week 2- Acne. Case Summary. Skin- Functions. Communication Heat regulation Excretion Absorption Protection Aesthetics Storage and synthesis Sensation CHEAPASS. Skin Anatomy. Skin Histology. Acute Inflammation. Outcomes of acute inflammation. Acne. Abscess. Microbiology.

273 views • 17 slides

Pediatric Infectious Obstructive Airway Diseases

Pediatric Infectious Obstructive Airway Diseases. Fred Hill, MA, RRT. Obstructive Airways Diseases of Children. Epiglottitis Croup Bronchiolitis. Epiglottitis: Etiology and Incidence. Acute inflammation and edema of supraglottic structures

290 views • 14 slides

Fate of Macrophages in inflammation

Fate of Macrophages in inflammation. After the initiating stimulus is eliminated and the inflammatory reaction abates, macrophages eventually die or wander off into lymphatics . In chronic inflammatory sites, however, macrophage accumulation persists by: 1- macrophages can proliferate .

805 views • 14 slides

Chronic Inflammation

Chronic Inflammation. Dr. Raid Jastania. Chronic Inflammation:. Chronic inflammation is a prolonged inflammation with continuous: Cell injury Active inflammation Ongoing healing (repair) process It is characterized by: Mononulcear cells (lymphocytes, macrophages, plasma cells)

1.91k views • 14 slides

Erythromycin: Schedule V Drug

Erythromycin: Schedule V Drug. By: Sam Bennett. http://www.youtube.com/watch?v=YBWuqL9o44I. History. Macrolide antibiotic, which slow the growth of, or sometimes kill, sensitive bacteria by reducing the production of important proteins needed by the bacteria to survive

379 views • 11 slides

Day Two Notes

Day Two Notes. Greater Omentum, Mesentery & Mesocolon. Lesser Omentum. Peritonitis. Acute inflammation of the peritoneum Cause contamination by infectious microbes during surgery or from rupture of abdominal organs. Mouth.

179 views • 9 slides

Sections 5 Course & outcome of Inflammation

Sections 5 Course & outcome of Inflammation. Course. (1) Acute inflammation (2) Chronic inflammation. Outcomes. (1) Healing (2) Delay and Persistence (3) Spread ? Direct ? Lymphatic: Lymphangitis progressing to acute lymphadenitis. ? Blood vessels Bacteria: Organisms? blood

207 views • 5 slides

Abrupt weaning

Natural weaning. Abrupt weaning. Involution stage II. Involution stage I. Histology of involuted gland. Histology of lactating gland. The first stage of weaning is characterized by acute inflammation.. The alveoli is packed with large number of neutrophils. Involution stage I.

100 views • 1 slides