Stanley foundation neuropathology consortium - PowerPoint PPT Presentation
Center for Translational Neuroscience Seminar Series
Center for Translational Neuroscience Seminar Series. Tuesday , July 9, 12 noon Rayford Auditorium, Biomed II Bldg. “ Review of: Up -regulation of neuronal calcium sensor-1 (NCS1) in the prefrontal cortex of schizophrenic and bipolar patients ” Stasia D’Onofrio
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NEUROPATHOLOGY
NEUROPATHOLOGY . Microscopic image. Q9. List three key histopathologic features as noted at the arrows. Large pleomorphic cells, angiogenesis, necrosis Q10. What is the diagnosis? Glioblastoma. Q7. Is this benign or malignant? Why? .
Neuropathology
Neuropathology. Strategies for conducting research with postmortem human brains. What neuropathologists do!. Perform autopsies to examine : CNS [brain, spinal cord]; PNS [peripheral nerves, end-organ nerves]; Skeletal muscle; Eyes; Pituitary gland
Neuropathology I
Neuropathology I. Main elements of the CNS and their basic pathologic reactions. Neurons marked variations in size and shape (Purkinje cell, granular cell) cell body + processes (axon and dendrites) Nissl substance (GER) – proteosynthesis hypoxic/ischemic injury
FACIAL NEUROPATHOLOGY
FACIAL NEUROPATHOLOGY. OROFACIAL PAIN. NOCICEPTORS:1-5MM DIAMETER NERVE FIBERS. A-DELTA: Responsible for temperature and fast or first pain. Is myelinated Conduction velocity is 12-45 m/sec. C-FIBERS: Responsible for slow or second pain, and temperature. Is unmyelinated
Neuropathology III
Neuropathology III. Demyelinating diseases. Demyelinating diseases loss of myelin → c hanged ability of axons to transmit signals → various neurological deficits white matter affected Multiple sclerosis most common demyelinating disorder (1/1000 persons)
Developmental Neuropathology
Developmental Neuropathology. Pathology, Radiology, and Clinical Correlations Bob Hevner, MD, PhD University of Washington Harborview Medical Center. Salvador Dali. Classification & Overview. Malformations
Neuropathology II
Neuropathology II. Vascular brain diseases. Global cerebral ischaemia (hypoxic encephalopathy) decrease of cerebral perfusion pressure below 45mmHg cardiac arrest, shock, severe hypoxia various sensitivity to hypoxia: neurons > glial cells
Neuropathology Conference
Neuropathology Conference. 9/18/2017 Xiaowei (Bill) Su, aka Red Panda , PGY-2 Dr. Julia Kofler. Case 1. CC: OSH transfer for (?)acute/subacute stroke w/ hemorrhagic conversion HPI: 80-yo F, mRS 0, at prior baseline until morning of admission
Neuropathology III
Neuropathology III. Demyelinating diseases. Demyelinating diseases loss of myelin → c hanged ability of axons to transmit signals → various neurological deficits white matter affected Multiple sclerosis most common demyelinating disorder (1/1000 persons)