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FUNCTION TRANSVERSUS ABDOMINUS

FUNCTION TRANSVERSUS ABDOMINUS. SUPPORT OF ABDOMINAL CONTENTS VIA CIRCUMFERENTIAL ARRANGEMENT BILATERAL CONTRACTION CAUSES DRAWING IN OF ABDOMINAL WALL CAN WORK WITH MULTIFIDUS VIA TENSION OF THORACOLUMBAR FASCIA CONTRIBUTES TO BOTH SUPPORTING AND TORQUE ROLES. MULTIFIDUS. Multifidus.

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FUNCTION TRANSVERSUS ABDOMINUS

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  1. FUNCTION TRANSVERSUS ABDOMINUS • SUPPORT OF ABDOMINAL CONTENTS VIA CIRCUMFERENTIAL ARRANGEMENT • BILATERAL CONTRACTION CAUSES DRAWING IN OF ABDOMINAL WALL • CAN WORK WITH MULTIFIDUS VIA TENSION OF THORACOLUMBAR FASCIA • CONTRIBUTES TO BOTH SUPPORTING AND TORQUE ROLES

  2. MULTIFIDUS

  3. Multifidus

  4. FUNCTION (MULTIFIDUS) • Provides control of shearing forces of intervertebral motion segments • Unique segmental arrangement of multifidus suggests capacity for fine control of movement • Control anterior rotation translation in trunk flexion • Continuously active in upright posture compared with recumbency • Provides anti gravity support • Active in both ipsilateral and controlateral trunk rotation • Stabiliser rather than prime mover

  5. Gluteal Stabilizers

  6. Hip Musculature • Psoas • Closed chain vs. open chain functioning • Works with erector spinae, multifidus & deep abdominal wall • Works to balance anterior shear forces of lumbar spine • Can reciprocally inhibit gluteus maximus, multifidus, deep erector spinae, internal oblique& transverse abdominus when tight • Extensor mechanism dysfunction • Synergistic dominance during hip extension • Hamstrings & superficial erector spinae • May alter gluteus maximus function, altering hip rotation, gait cycle

  7. Gluteus medius: provides frontal plane stabilization, decelerate femoral adduction , assist in deceleration femoral internal rotation (during closed chain activity)

  8. Gluteus Medius • Provides frontal plane stabilisation in walking cycle • Prevents downward rotation of the pelvis (Trendelenburg) • Allows unsupported leg to swing clear of the ground • Decelerates femoral adduction and internal rotation • Anterior fibres assist the iliotibial tract to flex hip and stabilise the extended knee

  9. Hip Musculature • Gluteus medius • Frontal plane stabilizer • Weakness increases frontal & transverse plane stresses (patellofemoral stress) • Controls femoral adduction & internal rotation • Weakness results in synergistic dominance of TFL & quadratus lumborum • Gluteus maximus • Hip extension & external rotation during OKC, concentrically • Eccentrically hip flexion & internal rotation • Decelerates tibial internal rotation with TFL • Stabilizes SI joint • Faulty firing results in decreased pelvic stability & neuromuscular control

  10. Hamstrings • Concentrically flex the knee, extend the hip & rotate the tibia • Eccentrically decelerate knee extension, hip flexion & tibial rotation • Work synergistically with the ACL to stabilize tibial translation • All muscles produce & control forces in multiple planes

  11. Neuromuscular efficiency • Ability of CNS to allow agonists, antagonists, synergists, stabilizers & neutralizers to work efficiently & interdependently • Established by combination of postural alignment & stability strength • Optimizes body’s ability to generate & adapt to forces • Dynamic stabilization is critical for optimal neuromuscular efficiency • Rehab generally focuses on isolated single plane strength gains in single muscles • Functional activities are multi-planar requiring acceleration & stabilization • Inefficiency results in body’s inability to respond to demands • Can result in repetitive microtrauma, faulty biomechanics & injury • Compensatory actions result

  12. The CORE • Functions & operates as an integrated unit • Entire kinetic chain operates synergistically to produce force, reduce force & dynamically stabilize against abnormal force • In an efficient state, the CORE enables each of the structural components to operate optimally through: • Distribution of weight • Absorption of force • Transfer of ground reaction forces • Requires training for optimal functioning! • Train entire kinetic chain on all levels in all planes

  13. Core Stabilization Concepts • A specific core strengthening program can: • IMPROVE dynamic postural control • Ensure appropriate muscular balance & joint arthrokinematics in the lumbo-pelvic-hip complex • Allow for expression of dynamic functional performance throughout the entire kinetic chain • Increase neuromuscular efficiency throughout the entire body • Spinal stabilization • Must effectively utilize strength, power, neuromuscular control & endurance of the “prime movers” • Weak core = decreased force production & efficiency • Protective mechanism for the spine • Facilitates balanced muscular functioning of the entire kinetic chain • Enhances neuromuscular control to provide a more efficient body positioning

  14. Postural Considerations • Core functions to maintain postural alignment & dynamic postural equilibrium • Optimal alignment = optimal functional training and rehabilitation • Segmental deficit results in predictable dysfunction • Serial distortion patterns • Structural integrity of body is compromised due to malalignment • Abnormal forces are distributed above and below misaligned segment

  15. Neuromuscular Considerations • Enhance dynamic postural control with strong stable core • Kinetic chain imbalances = deficient neuromuscular control • Impact of low back pain on neuromuscular control • Joint/ligament injury  neuromuscular deficits • Arthrokinetic reflex • Reflexes mediated by joint receptor activity • Altered arthrokinetic reflex can result in arthrogenic muscle inhibition • Disrupted muscle function due to altered joint functioning

  16. Optimum Dynamic Function Integrated proprioceptively enriched multi-directional movement controlled by an efficient neuromuscular system

  17. PROPRIOCEPTION “Nerve impulses originating from the joints, muscles, tendons and associated deep tissues which are then processed in the central nervous system to provide information about joint position, motion, vibration and pressure”. (Bruckner & Khan 1999)

  18. WHY IS PROPRIOCEPTION IMPORTANT? • Sub-cortical systems are not under conscious control • Stabilization response needs to be second nature. • Sub-cortical systems act faster - rapid muscle reaction times. • More rapid reaction times can be learnt which may lead to increased stability of the lumbar spine.

  19. To improvethe proprioceptive system in dynamic joint stability it must be challenged. • Pain-free does not mean cured. • If the proprioceptive deficit has not been addressed a complete rehabilitation has not been accomplished. • Mechanically stable joints are not necessarily functionally stable ( eg. ACL reconstruction)

  20. WHAT HAPPENS WHEN THE SYSTEM GOES WRONG? The Theories

  21. “MUSCLE PAIN SYNDROMES ARE SELDOM CAUSED BY ISOLATED PRECITATING FACTORS AND EVENTS BUT ARE THE CONSEQUENCES OF HABITUAL IMBALANCES IN THE MOVEMENT SYSTEM” (Sahrmann 1993)

  22. REPEATED MOVEMENTSSUSTAINED POSTURES • ALTERS MUSCLE LENGTH • ALTERS STRENGTH • ALTERS STIFFNESS • ALTERS FLEXIBILITY • ALTERS CARTILAGE AND BONE STRUCTURE – BY OVERLOADING AT COMPENSATORY SITES OF MOVEMENT

  23. PAIN MUSCULAR DYSFUNCTION POSTURAL DYSFUNCTION STRUCTURAL/SEGMENTAL DYSFUNCTION

  24. POSTURE AND PAIN • Poor posture can lead to increased stress on the stabilising system of the joints (Chek P 1999) • Multifidus dysfunction occurs after first episode acute unilateral LBP (Hides et al 1994) • Multifidus dysfunction does not spontaneously restore following resolution of pain and disability (Hides et al 1996) • Specific retraining does restore dysfunction (Hides et al 1996)

  25. TrA contraction is delayed during normal movements in subjects with low back pain (Richardson et al 1999) • Mulifidus function can be affected by spinal surgery • Atrophy of multifidus has been shown to be more prevalent in post operative patients (Jull, et al 1999)

  26. Sherington’s Law of Reciprocal Inhibition: • Tight Muscles inhibit the functional antagonist. • Leads to Positive Cross Syndromes of the lower or upper limb

  27. Gluteus Maximus and minimus are inhibited in most athletes due to tight psoas (Summer, 1988).

  28. Poor recruitment in the local stabilisers can lead to over- activity of the global stabilisers to compensate.

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