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ACUTE ABDOMEN. Begashaw M . ACUTE ABDOMEN. Denotes any sudden condition with chief manifestation of pain of recent onset in the abdominal area which may require urgent surgical intervention. Sites of referred pain . Sites of Abdominal Pain . CLASSIFICATION. Obstruction Inflammation
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ACUTE ABDOMEN Begashaw M
ACUTE ABDOMEN • Denotes any sudden condition with chief manifestation of pain of recent onset in the abdominal area which may require urgent surgical intervention
CLASSIFICATION • Obstruction • Inflammation • Hemorrhage • Infarction • perforation
CLINICAL FEATURES • Symptoms _Colicky and Intermittent pain ( visceral) _Continuous pain ( somatic) _Vomiting _Fever _Tachycardia • Colic pain obstruction • Continuous paininfection, inflammation or ischemia
Signs • Abdominal distention, visible peristalsis • Direct and rebound tenderness, guarding • Anemia, hypotension • Toxic with Hippocratic faces • Absence of bowel sound ( peritonitis) • Psoassignappendicitis • Murphy‘s signacutecholecystitis • Dehydrationsunken eyeballs
DIFFERENTIAL DIAGNOSIS • Surgical - Intestinal obstruction • Gynecologic & obstetric - Ectopic ruptured pregnancy • Medical - enteritis
Surgical causes A- InflammationAcute appendicitis Acute cholecystitis B- Obstruction Intestinal obstruction C- Infarction Mesenteric ischemia D-Strangulation volvulus E- Perforation perforated peptic ulcer
DIAGNOSIS Clinical: Hx & p/E Lab: CBC, cross match, urine analysis, serum amylase & electrolytes Ultrasound plain film of abdomen
MANAGEMENT A-Preoperative - Resuscitation with IV fluids - Antibiotics - Catheterization & NGT insertion - Analgesics after confirming the diagnosis B- Surgery Definitive laparotomy CMonitoring Follow up
INTESTINAL OBSTRUCTION • is partial or complete blockage of the intestine producing symptoms _Vomiting _Constipation _Distension _Abdominal pain
CLASSIFICATION Mechanical physical barrier blocks Paralytic ileusdisordered propulsive motility High _Small bowel Low _Large bowel Simple -> adequate blood supply Strangulated -> Mesenteric vessels occluded
Mechanical A- Luminal _Gallstone Ileus _Food bolus _MeconiumIleus _Malignancy _Inflammatory mass _Ascaris bolus B- Mural _Stricture _Congenital _Inflammatory _Ischemic _Neoplastic _Intussusception
C- Extra mural Adhesionsinflammatory/malignant Hernia External/internal VolvulusSmall bowel large bowel -> Sigmoid volvulus
PATHOPHYSIOLGY • Proximal dilatation disrupts peristalsis • Above the obstruction distended with fluid and gas • stimulates excessive peristalsis ->colicky pain • blood vessels-stretched & narrowed ischemia • Absorptive capacity decreases • increased vomiting depletion of extra cellular fluid hypovolemia & dehydration
A strangulated loop dies and perforates to produce severe bacterial peritonitis which is often fatal Grossly distended abdomen restricts diaphragmatic movement and interferes with respiration A multiple organ failure
Clinical features • Symptoms -Abdominal pain-colic -Vomiting -Constipatio-partial -absolute • Signs -Abdominal distension visible bowel loops -High pitched bowel sounds -Tenderness & guarding -Dehydration & hypotension -Empty rectum DRE Large bowel obstruction
DIAGNOSIS • Clinical: Hx & P/E • Lab: CBC, electrolytes • Plain abdominal film : - distension of bowel with air fluid level - Central located distended loops with multiple air fluid levelsmall bowel - Peripherally located distended bowel with haustralmarksLarge bowel
MANAGEMENT Fluids resuscitation to restore the circulatory state Early preoperative preparation Attempt rectal tube deflation-simple sigmoid volvulus Supportive measures Early operationLaparotomy Post operative care
SIGMOID VOLVULUS • Sigmoid colon is the most frequent site of volvulus • Predisposing factors - A long redundant sigmoid with a narrow pedicle - High fiber diet • Chronic constipation_elderly _chronic mental pts
PATHOPHYSIOLOGY Redundant sigmoid twists on its base in a clockwise direction Mesocolic veins become occluded & arterial inflow into the twisted loop perpetuates the volvulus until it becomes irreversible Twisted loop distends grossly Perforation may occur due to either pressure necrosis at the base of the twist or to avascular necrosis at the apex
DIAGNOSIS • CLINICAL _Abdominal cramp & distension _Constipation (early) & vomiting (late) _Empty rectum on DRE • RADIOLOGIC FINDINGS • Two long fluid levels in the lower quadrant • Inverted U shape of the sigmoid lumen • “Coffee bean” appearance or the ‘Omega sign”
MANAGEMENT • Conservative • simple volvulusdeflation with a well greased large bore rectal tube under the guide of a sigmoidoscope • Deflation fails laparotomy & derotation • Elective resection & anastomosis • Intravenous fluid - rehydrate if sign of dehydration
Emergency Surgery _Complicated volvulus with signs of peritonitis _Resuscitative measures _Antibiotics _Resection of the gangrenous segment with Hartman’s colostomy
APPENDICITIS is an inflammation of the appendix that results from bacterial invasion usually distal to an obstruction of the lumen
Pathogenesis • Luminal obstruction bacterial overgrowth lnflammation/swelling Increased pressure-localized ischemiagangrene/perforationlocalized abscess (walled off by Omentum) or Peritonitis • Etiology: _Hyperplasia of lymphoid follicles _Fecolith, obstructing neoplasm _Parasites, foreign body
CLINICAL PRESENTATION Symptoms -Central abdominal colic which shifts to the right Iliac fossa -Anorexia, nausea, episodes of vomiting and low grade fever -High grade fever indicates perforation and peritonitis
Signs -Tenderness and localized rigidity in RLQ MC Burney’s point -Rovsing’s sign: Pain in RLQ on pressing in LLQ -Psoas sign: Pain on extension of right flexed hip -Obturator sign: Pain on passive internal or external rotation of the flexed right hip -Right sided tenderness on rectal examination. -Diminished bowel sounds indicating peritonitis
Differential diagnosis • IN CHILDREN -Intussusceptions -Mesenteric adenitis • FEMALE -PID -Twisted ovarian cyst( torsion) - ruptured ovarian follicle • GENERAL -Acute chlolecystitis -Perforated PUD -Renal or ureteric calculi -UTI -Early small bowel obstruction (volvulus) -Gastroenteritis
Investigations • Labs • leukocytosis with left shift • beta-hCG to rule out ectopic pregnancy • Urinalysis • Imaging: • Upright CXR, AXR-free air • Ultrasound: may visualize appendix
MANAGEMENT • PREOPERATIVE -Resuscitation with fluids -Appropriate antibiotics (combination for coverage of gram positive, gram negative and anaerobes) -Correct all deficits ( dehydration) • SURGERY -Surgical removal of the appendix is the definitive treatment-Appendectomy
COMPLICATIONS Perforation - local or generalized peritonitis Appendiceal mass and abscess formation Death