1 / 46

Eviscerations

Eviscerations. Why eviscerate?. Scleral shell left behind –better cosmesis. Indications: blind painful..painless / disfigured / not responding to treatment eye scenarios. Comprise of blood frags, corneo-scleral disc, retina and uveal tissue, lens (lens prosthesis), vitreous.

lsuhr
Download Presentation

Eviscerations

An Image/Link below is provided (as is) to download presentation Download Policy: Content on the Website is provided to you AS IS for your information and personal use and may not be sold / licensed / shared on other websites without getting consent from its author. Content is provided to you AS IS for your information and personal use only. Download presentation by click this link. While downloading, if for some reason you are not able to download a presentation, the publisher may have deleted the file from their server. During download, if you can't get a presentation, the file might be deleted by the publisher.

E N D

Presentation Transcript

  1. Eviscerations

  2. Why eviscerate? • Scleral shell left behind –better cosmesis. • Indications: blind painful..painless / disfigured / not responding to treatment eye scenarios. • Comprise of blood frags, corneo-scleral disc, retina and uveal tissue, lens (lens prosthesis), vitreous.

  3. What do you do with them? • Examine all fragments. • Bisect tissues • H&E, PAS, Perls +/- bug stains. • Are there histological hallmarks of sympathetic ophthalmia. • Any tumour? • Any pathology that can inform how the other eye will do?

  4. Band keratopathy Dystrophic calcification

  5. Acute inflammation

  6. Chronic inflammation-chronic keratitis Blood vessels Plasma cells

  7. Infective agent-bacteria

  8. Endothelial Cell Loss

  9. Host-donor interface scar

  10. New vessels on anterior stromal surface

  11. Ectropion uveae, secondary to rubeosis

  12. Grossly thickened basement membrane of pars plicata

  13. Brown pigment clogging trabecular meshwork

  14. Proliferative diabetic retinopathy Note new-vessels, surrounded Fibrous tissue on retinal surface.

  15. Normal retinal arteriole

  16. Arteriolosclerosis-note thick wall-called hyalinisation. Hyalinisation due to accumulation of lipid and fibrin, from leaky vessel, due to pericyte death

  17. Occluded branch venule. Note inflammation around vessel -attempts at organisation of thrombus

  18. Perl’s stain picks up haemosiderin Stains it intense blue. This indicates previous intra-retinal haemorrhage from BRVO

  19. Hard druse. Pink, homogeneous nodular material Lies between RPE basement membrane and inner collagenous part of Bruch’s. Note overlying RPE arophy

  20. Thickened Bruch’s Fluffier, more diffuse, soft drusen

  21. Thick Bruch’s, hard drusen and complete loss of photoreceptors

  22. Haemorrhage of CNVM. CNVM lies between neural retina And RPE Eventual organisation of haemorrhage Will lead to disciform scar. RPE choroid CNVM RPE RETINA

  23. Vessel of CNVM CNVM choroid RPE

  24. Choriocapillaris intact Granulomatous (non-caseating) inflammation of uvea (choroid in this case), sparing the choriocapillaries

  25. PRP SCAR

  26. Loss of choriocapillaris Loss of RPE Bruch’s Gliotic retina PRP SCAR-loss of RPE and choriocapillaris Retinochoroidal atrophy Retinal gliosis Gliotic retina in direct apposition to Bruch’s

More Related