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THE ENG BATTERY

THE ENG BATTERY. ENG & VNG. Clinical Eye Movement Videos. Calibration. Confirming relation between: Voltage/Infrared video feed and Eye position Fixed Targets/Sinusoidal Tracking. Gaze testing. Gaze at visual targets. Eye movements are recorded Spontaneous nystagmus

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THE ENG BATTERY

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  1. THE ENG BATTERY

  2. ENG & VNG Clinical Eye Movement Videos

  3. Calibration • Confirming relation between: • Voltage/Infrared video feed and • Eye position • Fixed Targets/Sinusoidal Tracking

  4. Gaze testing • Gaze at visual targets. • Eye movements are recorded • Spontaneous nystagmus • gaze evoked nystagmus • other extraneous movments • Pt. asked to close there eyes without shifting gaze.

  5. strongest on gaze in direction of beating never vertical declines quickly (within days to a couple of weeks) Alexander's Law:1st degree Nystagmus: present only on lat. gaze2nd deg: both on center and lat. side of beat3rd deg: on center, and both lateral gazes. Video Periph Gaze Peripheral Gaze Nystagmus:

  6. Alexander's Law

  7. Central Nervous System Lesions: • Often bilateral beating • Can have vertical beating • declines slowly if at all

  8. Some Central Gaze Nystagmi: • Bilateral Horiz. Gaze (Brun's) Nystagmus: • Rebound Nystagmus: • Periodic Alternating Nystagmus: • Vertical Nystagmus: • Congenital Nystagmus: What is Going on here?:Voluntary Nystagmus

  9. Bilateral Horiz. Gaze (Brun's) Nystagmus: • in large CPA tumors. • Gaze ipsi to lesion generates large slow nyst, with exp. decay in slow phase. • Gaze contra to lesion generates small fast nyst, in opposite direction of ipsi resp. • Video Bruns

  10. Rebound Nystagmus: • Cerebellar disease • movement-generated, decays rapidly (10-20s) • Beats in direction of movement • Video Rebound

  11. Periodic Alternating Nystagmus: • Medullary disease. Periodic Alternating Video • cyclic, 90 s one direction, • 10 s nothing or vertical, • then 90s in other direction, 10 s down time, • and back again. • present w/ eyes open or closed. • strongest in middle of phases>>visual impairment.

  12. Vertical Nystagmus: • Brainstem/Cerebellar or Inf. olivary disease • Can be generated by alcohol, drugs, too. • Upbeat Video • Downbeat Video

  13. Congenital Nystagmus: • From fixed brain defect either genetic or developmental in origin. • Pendular and/or jerk-type • Disorder of slow eye movement sub-system. • Null points or periods. • Convergence inhibition • Congenital Video

  14. Saccade Testing • Horizontal • Vertical • Regular pattern or random • Through 20 to 30 degrees. 

  15. Saccadic Disorders: • Occular dysmetria: CBL lesion • akin to dysdiadochokinesia • overshoots/undershoots • Saccadic Slowing: basal ganglia lesion • normal saccade for 20 deg = 188/sec • Internuclear Ophthalmoplegia: MLF lesion • rounded tracings • one eye lags, smoothing curve. • separate eye recordings to confirm INO VIDEO

  16. Watch out for: • Superimposed nystagmii) gaze nystagmusii) congenital nystagmus • Drug effects: usually dysmetria • Patient problems:i) inattentionii) eye blinksiii) head movement: scalloped tracings

  17. Tracking Tests: • Following pendular movements • Problems to look for • saccadic pursuit-eyes snap repeatedly to keep up with movement = CNS lesion • disorganized pursuit, wandering, slow, inaccurate tracking - CNS lesion, usually above the level of theocculomotor nuclei • disconjugate pursuit, eyes don't stay together in tracking - CNS lesion

  18. Things to look out for: • Drug influences • Inattention: multiple, rapid gaze deviations • Head movement: depressed amplitude • superimposed nystagmus • gaze: R, L, or bil. >> jerks at extremes • congenital: often overlies entire tracing

  19. Optokinetic test • Repeated tracking of moving target, producing nystagmatic motion. • Disorders: • Asymmetry: CNS lesiondiff of > 30 degs, at more than one stim rate. • Flat / declining resp. to faster rates. brainstem lesion, possible MS • Inverted movement: Congenital nystagmus

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