Vascular Surgery Back to Basics - PowerPoint PPT Presentation

vascular surgery back to basics n.
Skip this Video
Loading SlideShow in 5 Seconds..
Vascular Surgery Back to Basics PowerPoint Presentation
Download Presentation
Vascular Surgery Back to Basics

play fullscreen
1 / 82
Vascular Surgery Back to Basics
Download Presentation
Download Presentation

Vascular Surgery Back to Basics

- - - - - - - - - - - - - - - - - - - - - - - - - - - E N D - - - - - - - - - - - - - - - - - - - - - - - - - - -
Presentation Transcript

  1. Vascular SurgeryBack to Basics Dr. Srinidhi Jayaram Vascular Fellow Division of Vascular & Endovascular Surgery The Ottawa Hospital

  2. OUTLINE • Acute limb ischemia • Claudication • Critical limb ischemia • Carotid Artery Disease • Aortic Aneurysm • Aortic dissection • Varicose veins, Chronic venous insuffciency, Superficial thrombophlebitis

  3. Mrs. Witelegg • ID: • 75 yo lady who lives by herself in an apartment. She is active, walks her dog ~2 kms daily without any difficulty. She takes pride in the fact that she has not needed to see a doctor in the last 10 years. • PMHx/PMSx: • remote TAH-BSO • social smoker quit in the 1960’s • no h/o DM, CAD, HTN, dyslipidemia, stroke, CRF Acute Limb Ischemia

  4. Mrs. Witelegg • HPI: • While watching TV, she had sudden onset of numbness in her right leg. Her leg felt “like it went dead”, and she couldn’t ambulate. After a few minutes she experienced constant, severe pain starting in the toes, eventually involving the entire right leg. She called her neighbor and then brought her in to the Civic emergency department. Acute Limb Ischemia

  5. Mrs. Witelegg • What is Acute limb Ischemia? • An abrupt cessation of arterial blood flow to an extremity resulting in hypoperfusion of tissue, threatening limb viability Acute Limb Ischemia

  6. Mrs. Witelegg • O/E • She is in distress from pain in R leg • BP:140/90 mmHg, HR:150 bpm • pulse: irregularly irregular • Normal heart sounds, good a/e bilat • No pulsatile masses in her abdomen • No carotid, abdominal or femoral bruits • Pulses: • L: + femoral, + politeal, + DP, + PT • R: - femoral, - popliteal, - DP, - PT • R foot is colder and paler than L • Decreased sensation in R foot • Able to move toes but difficulty with plantar and dorsi flexion • Absence of trophic changes in her lower extremities (no hair loss, thickened nails, or thin, flaky or shiny skin) Acute Limb Ischemia

  7. Mrs. Witelegg • What are the 6 P’s of Acute Limb Ischemia • Pain • Palor • Polar/poikilothermia • Paraesthesia • Paralysis • Pulselessness Acute Limb Ischemia

  8. Mrs. Witelegg • Classify Acute Limb Ischemia. In which category is Mrs. Witelegg? Acute Limb Ischemia

  9. Mrs. Witelegg • Your working diagnosis is acute limb ischemia. • You order CBC, electrolytes, BUN, Cr, PTT/INR (all of which comes back normal), type and cross-match blood, and a saline infusion is started. • CXR is unremarkable • ECG is as follows: Acute Limb Ischemia

  10. Mrs. Witelegg • Before you call the vascular surgeon on-call, what test can you do at the bed side that can objectively assess acute limb ischemia? • Ankle Brachial index • Measure brachial pressure (example 160 mmHg) • Measure ankle pressure (example: 80 mmHg) • Divide ankle by brachial pressure (exmple 80/160 = 0.5; anything <0.9 is abnormal) Acute Limb Ischemia

  11. Mrs. Witelegg • What is the most likely etiology of ALI in Mrs. Witelegg? • Cardiogenic embolism • What in her history and physical supports this diagnosis? • Lack of atherosclerotic risk factors • no previous claudication (she walked her dog 2 km/day) • Irregularly irregular pulse • Completely normal left extremity pulses • Based on her physical examination, what is the highest point of obstruction of arterial flow? • R ileo-femoral region • How long can a limb be without blood flow before irreversible tissue damage ensues? • 4-6 hrs Acute Limb Ischemia

  12. Mrs. Witelegg • What is the surgical management of this condition? • R femoral embolectomy • Can we proceed to the OR without any imaging studies? If not what studies can be perfomed? • Because of the classic history and physical findings, and because of the presence of class 2b ischemia, immediate surgery is indicated without delay for imaging. • Angiography can be performed in certain conditions of ALI • when the suspected etiology is arterial thrombosis (i.e. in preparation for bypass surgery) • when the patient has class 1 or 2a ischemia Acute Limb Ischemia

  13. Mrs. Witelegg • What medical therapy is available for ALI and when is it indicated? • Lytic therapy (i.e. with t-PA) is used to dissolve the clot. It is a good option in the setting of acute arterial or graft thrombosis. It is not indicated in the setting of trauma or when the patient can not wait more than 24-48 hrs, as the therapy requires that period of time for clot dissolution. ( i.e. class 1 or early 2a ischemia) • IV Heparin will not dissolve the clot but will prevent further propagation, and is only indicated if there is a delay to surgery

  14. Mrs. Witelegg • The patient is booked for emergency embolectomy • Under local anaesthesia, a small incision is made over the R groin. The femoral artery is exposed and controlled with vessel loops. A small arteriotomy is made and the clot is removed proximally and distally using a fogarty balloon embolectomy catheter. • The arteriotomy is repaired and the foot “pinks up” after blood flow is returned. There is a palpable DP and PT pulse. • The patient is returned to the recovery room. Acute Limb Ischemia

  15. Mrs. Witelegg • At 3 am you get paged by the recovery room nurse. Mrs. W is complaining of significant pain in her leg, it is more swollen and the DP and PT are no longer palpable. • In addition, her urine output has diminished and she is peeing out dark urine which tested positive for “blood” on the urine dipstick. Acute Limb Ischemia

  16. Mrs. Witelegg • What is happening to Mrs. W? • Reperfusion syndrome: occurs as a result of blood flow going back into previously damaged tissue, causing rhabdomyolysis and compartment syndrome.. • Rhabdomyolysis: Liberated myoglobin from dead muscle cells enters the blood stream resulting in renal tubular obstruction and direct nephrotoxicity causing renal failure. Myoglobinuria is a false positive on the urine dipstick test for blood. • Compartment syndrome: Free oxygen radicals are created with reperfusion. These result in increased tissue edema, with in the limited facial compartments of the lower leg, this further decreases capillary blood flow and worsens the ischemia and tissue damage, causing further edema. Pain out of proportion, pain on passive stretch and high pressures in the compartments suggests compartment syndrome. Acute Limb Ischemia

  17. Mrs. Witelegg • How should reperfusion syndrome be managed? • Compartment syndrome is a surgical emergency and is managed by 4-compartment fasciotomies. • Rhabdomyolysis should be managed with aggressive IV fluids, diuresis and alkalinization of urine. Acute Limb Ischemia

  18. Peripheral Vascular Disease

  19. Peripheral Vascular Disease • Claudication • Critical Limb Ischemic • Rest pain • Ulcers • Gangrene

  20. Etiology • blockages in arteries to lower extremities due to atherosclerosis • Risk factors • smoking • DM • HTN • hyperlipidemia • family history • obesity • sedentary • male gender

  21. Clinical Features - Claudication • Pain with exertion (usually calves) • relieved by short rest - two to five minutes • reproducible • P/E • hair loss, hypertrophic nails, atrophic muscle • pulses may be absent at some locations

  22. Investigations • Ankle Brachial Index • Duplex • Ct angio/ Angiogram

  23. Treatment • CONSERVATIVE • risk factor modification ( smoking cessation) • exercise program • cilostazol , Trental (Pentoxifylline) • anti platelet (ECASA, clopidrogel) for MI / stroke risk • Statin • ACE inhibitor • surgical • indications: claudication interfering with lifestyle • options: endovascular, PTA, arterial bypass grafts


  25. Clinical Features - Critical limb ischemia • Pain at rest in foot, worse at night • Improved with dependant postion • Ischemic ulcers • gangrene • P/E • Pulse deficits • hair loss, hypertrophic nails, atrophic muscle • ruborous foot

  26. Investigations • Ankle Brachial Index • Duplex ultrasound • Ct angio/Angiogram

  27. Treatment • Surgical • bypass • gortex vs vein • Endovascular balloon angioplasty • limited durability • less morbid

  28. Cerebrovascular Disease

  29. CAROTID STENOSIS: Presentation: • Asymptomatic: • Bruit (only 20% hemodynamically significant lesion) • Screening prior to other surgery

  30. Presentation: • Symptomatic: • TIA, Stroke • Amaurosis fugax ipsilateral to carotid lesion • Contralateral motor or sensory deficit • Facial droop • Dysphasia or aphasia

  31. Investigations • Duplex Scan • CT scan - confirm or r/o infarct • CT/Angio – Confirm U/S plan • MRA – Similar to CT • Angiogram

  32. Management • Asymptomatic - Risk factor reduction(asa,statin,ACE) • observation with regular duplex scans • Antiplatelet agent and surgery more controversial • ACAS  60%  OR • Canada  80% male, under 75 yrs or  operate

  33. Symptomatic: Carotid Stenosis  70% TIA, Small completed stroke with minimal residual neurologic deficit,  antiplatelet agent +carotid endarterectomy

  34. AORTIC DISSECTION Vascular Surgery – Back to Basics

  35. Definition • spontaneous tear in aortic intima allowing blood to be driven between the aortic intima and media • acute < 2 weeks • chronic > 2 weeks

  36. Classification • DeBakey • Type I - involves ascending and descending aorta • Type II - ascending aorta only • Type IIIA - descending thoracic aorta • Type IIIB - Type IIIA plus abdominal aorta • Standford • Type A - ascending aorta and aortic arch; emergency • Type B - aorta distal to subclavian artery; emergency surgery if complications of dissection

  37. Etiology • HYPERTENSION, usually uncontrolled • TRAUMA, usually deceleration injury (falls, MVAs) • other: cystic medial necrosis, atherosclerosis, connective tissue disease (Marfan’s syndrome, Ehlers-Danlos syndromes), congenital conditions (coarctation of aorta, bicuspid aortic valves, PDA), infection, arteritis (Takayasu’s)

  38. Epidemiology • incidence 5.2 in 1,000,000 • male:female = 3:1 • small increased incidence in African-Canadians (related to higher incidence of hypertension) • lowest incidence in Asians

  39. Clinical Features • SUDDEN ONSET SEVERE CHEST PAIN RADIATION TO THE BACK (INTERSCAPULAR) +/-.... • hypertension • asymmetric BP’s and pulses between arms • ischemic syndromes due to occlusion of aortic branches: coronary (MI), carotid (stroke, Horner’s syndrome), splanchnic (ischemic gut), renal (kidney failure) • “unseating” of aortic valve cusps (new diastolic murmur) • rupture into pleura (dyspnea, hemoptysis) or peritoneum (hypotension, shock) or pericardium (tamponade) • lower limb ischemia (cold legs)

  40. Investigations • CT scan is gold standard • CXR • pleural cap • widened mediastinum • left pleural effusion with extravasation of blood • TEE • ECG: LVH (90%), +/- MI, pericarditis, heart block • aortography, MRI

  41. Treatment • Type A • EMERGENCY CARDIAC SURGERY • may require putting patient on pump, hypothermic circulatory arrest, valve replacement, coronary re-implantation of aortic root • resection of intimal tear, reconstitution of flow through true lumen, replacement of the affected aorta with graft • Type B • MEDICAL MANAGEMENT • very rarely urgent operation for complications (expansion, rupture, gut/leg/renal ischemia, ongoing pain


  43. Definition • localized dilation of an artery that is 2 x its normal diameter • true aneurysm: involving all vessel wall layers • false aneurysm: disruption of aortic wall with containment of blood by some layers of the aorta or a fibrous capsule made of surrounding tissue

  44. Etiology • DEGENERATIVE (matrix metalloproteinases) • atherosclerosis association • infection • cystic medial necrosis • trauma • vascultitis • connective tissue disease (Marfan syndrome, Ehlers-Danlos)