Differentiating among incretin agents for type 2 diabetes weighing the evidence
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Differentiating Among Incretin Agents for Type 2 Diabetes: Weighing the Evidence. Program Goals. Insulin Deficiency and Glucagon Hypersecretion in T2D. Postprandial Glucagon Is Excessive and Not Corrected by Exogenous Insulin. Incretin Hormome Physiology. Signal Transduction and GLP-1.

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Presentation Transcript

Program goals
Program Goals Weighing the Evidence




Incretin hormome physiology
Incretin Hormome Physiology Exogenous Insulin











Liraglutide exenatide black box warning risk of thyroid c cell tumors
Liraglutide/Exenatide: Black Box Warning TimeRisk of Thyroid C-Cell Tumors


Incretin based therapies concern about acute pancreatitis
Incretin-based Therapies: TimeConcern About Acute Pancreatitis


Sitagliptin and exenatide associated with increased risk for hospitalization for acute pancreatitis
Sitagliptin and Exenatide Associated With Increased Risk for Hospitalization for Acute Pancreatitis


Incretin based therapies renal impairment
Incretin-based Therapies: Renal Impairment Hospitalization for Acute Pancreatitis




Closing comments
Closing Comments Alpha-Cell Hyperplasia


Abbreviations
Abbreviations Alpha-Cell Hyperplasia


Abbreviations cont
Abbreviations (cont) Alpha-Cell Hyperplasia


Abbreviations cont1
Abbreviations (cont) Alpha-Cell Hyperplasia


References
References Alpha-Cell Hyperplasia


References cont
References (cont) Alpha-Cell Hyperplasia


References cont1
References (cont) Alpha-Cell Hyperplasia


References cont2
References (cont) Alpha-Cell Hyperplasia


References cont3
References (cont) Alpha-Cell Hyperplasia


References cont4
References (cont) Alpha-Cell Hyperplasia