Mercury Biochemistry and its Relationship to Neurological Diseases Dr. Boyd Haley Professor Department of Chemistry University of Kentucky. May 2007. VISUALIZATION OF MERCURY EMITTING FROM A DENTAL AMALGAM. From: www. uninformed concent.com David Kennedy’s IAOMT tape
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Mercury Biochemistry and its Relationship to Neurological Diseases Dr. Boyd HaleyProfessor Department of ChemistryUniversity of Kentucky
IN SPITE OF THE OBVIOUS EMISSION OF Hg VAPORS FROM DENTAL AMALGAM THE FDA HAS STEADFASTLY REFUSED TO TEST THEM FOR SAFETY!
DeRouen et al. JAMA 295, 1784-92, 2006
LEVELS ng/g Hg Sb
Controls 8.0 1.5
IDCM 178,400 19.260
Frustaci et al., J. of American College of Cardiology, 33, (6) 1578, 1999. Controls were patients with valvular or ischemic heart disease.
ATHLETIC YOUTH DIE OF IDCM.
WHY HASN’T NIH REQUESTED PROPOSALS FOR RESEARCH TO STUDY THIS??
THIS IS PROOF THAT MERCURY CAN CONCENTRATE IN SPECIFIC TISSUES OR ORGANS OF THE BODY, EVEN IF Hg BLOOD LEVELS ARE FOUND TO BE IN THE NORMAL RANGE.
Normal Brain without and with Hg2+.
Alzheimer’s Disease Brain
With Disabilities Education Act (IDEA)
Thimerosal is toxic to tubulin and actin. Combinations of Hg2+ and thimerosal would be at least additive.
MOST VACCINES CONTAIN TRACES OF THIMEROSAL EVEN IF IT IS NOT ADDED AS A PRESERVATIVE.
The vaccine thimerosal concentration was (is) 125,000 to 250,000 nanomolar!
DR. MARK LOVELL’S LAB
Shubert et al. Combined Effects in Toxicology--A Rapid systematic Testing Procedure:Cadmium, Mercury & Lead. J. of Toxicology & Environmental Health 4:763, 1978.
“the administration of an essentially no response level (LD1) of a mercury salt together with a 1/20 of the LD1 of a lead salt killed all of the animals.” 2. “Generally, a combination was synergistic when the most toxic member was present at or near its LD1 dose in the presence of a much less toxic member.”
Conclusion: Mixing borderline toxic levels of two toxic metals (Pb2+ & Hg2+) makes an extremely toxic solution.
50 NANOMOLAR THIMEROSAL
DR. MARK LOVELL
Effects of Thimerosal on Nerve Growth Factor Signal Transduction and Cell Death in Neuroblastoma Cells. Parran et al., Toxicological Sciences, 2005.
Data demonstrated that thimerosal could alter NGF-induced signaling at concentrations lower than those causing neuronal death. Therefore, the neurons growth and properties could be impeded at exceptionally low levels of thimerosal without killing the neurons.
Estradiol Reduces Cumulative Mercury and Associated Disturbances in the Hypothalamus-Pituitary Axis of Ovariectomized Rats. Oliveria et al. Ecotoxicol. Environ. Safety Jan.10, 2006
Hair Hg level
Data from A. Holmes, M. Blaxill & B. Haley, Int. J. of Toxicology v22, 2003
Number of amalgams:
Control: autistic ratio:
Study1 Study2 Study3 Study4 Study5
*i.e., no increased risk of autism compared to low exposure group
After publication in 2005 all of the data for this work was “lost” by the CDC!!!
Go to Safeminds.org to read the FOIA material on the Verstraten studies.
Evidence of Harm
Conclusion; exposure to a potent neurotoxin, thimerosal, prevents autism!!! Nonsense!
It is my opinion that the CDC and FDA do not always have the interests of the USA citizens at the top of their list.
Make legislation at the state level that is more difficult to control by big pharmaceutical companies. Note that the FDA has ruled that their approval over ruled any state level legislation!
If you know a child with autism spectrum disorder inform the parents that autism is treatable. In many cases it is a form of early infant mercury toxicity.
Dr. Robert Nataf’s laboratory reports that many children diagnosed as “autistic” have prophyrin profiles indicating mercury toxicity. This is based on the research of Dr. James Wood of the University of Washington.
Get a urinary porphyrin profile done on your child to determine the possibility of mercury toxicity. Send the results to A-CHAMP.org. The compilation of these results will be presented to legislators throughout the USA.
RATES OF EACH ENZYME RX
IS DIFFERENTIALLY AFFECTED
BY Hg2+ VERSUS OTHER
RAPID BLOOD TO BRAIN MOVEMENT OF [203Hg]-THIMEROSAL:Gasset et al. Tetratogenicities of Opthalmic Drugs. Arch. Opthalomology 93, 52-55, 1975.
Stool Hg concentrationDaily Hg excretionDays to excrete X mcg
(ng/g) (mcg/day) (days) (days)
Minimum: 23 0.09-0.28 44.6-139670-2,083
Mean: 82 0.33-0.9812.7-37.9191-568
Maximum: 140 0.56-1.68 7.44-22.3112-335
As previously calculated, 11.4mcg of Hg were not accounted for after a single vaccination and had to be excreted or retained by body tissues within 48-72 hours, or 2-3 days.
As can be seen in the table above, it takes as a minimum about 7.4 days to excrete 12.5 mcg Hg using the maximum fecal Hg excretion rate. This includes an assumption of 1-3g feces/day/kg body weight.
Using the mean max daily excretion (0.98) then 1.96-2.94mcg of Hg of the 12.5mcg injected would be excreted leaving 10.54-9.56mcg or 84 to 76% Hg unaccounted for at days 2-3 respectively.
/-S-Hg-S-/ MUST BE LINEAR AND IT CANNOT BE WITH –SH GROUPS ON ADJACENT CARBONS.
WHAT FORMS IS THE EQUIVALENT OF DMSA-S-Hg-S-DMSA OR DMSA-Hg-CYSTEINE TYPE LINKAGES.
DMSA & DMPS WERE DEVELOPED IN THE 1940s!
Base compound binds Hg2+ tighter and more selectively than anything known to date.
Benzene bis-amido bis-thiol
Pyridine bis-amido bis-thiol
Water insoluble, but lipid soluble
Glutathione derivative of Functionalized Mercury Chelating Agents Making them Water Soluble
Note: Molecule would be charged and water soluble at pH 7.4.
Very water soluble