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High-Risk Births & Obstetric Emergencies. Chapter 36. INTRAPARTAL COMPLICATIONS. Interference with normal processes & patterns of labor/birth resulting in maternal or fetal jeopardy.

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intrapartal complications
INTRAPARTAL COMPLICATIONS
  • Interference with normal processes & patterns of labor/birth resulting in maternal or fetal jeopardy.
  • Preterm labor; dysfunctional labor patterns; prolonged labor; hemorrhage – uterine ruputure/inversion; amniotic-fluid embolus.
dysfunctional labor
Dysfunctional Labor:
  • Possible Causes:
    • Catecholamines (response to anxiety/fear), increase physical/psychological stress, leads to myometrial dysfunction; painful & ineffective labor.
    • Premature or excessive analgesia, particularly during latent phase.
    • Maternal factors.
    • Fetal factors.
    • Placental factors.
    • Physical restrictions (position in bed).
assessment
ASSESSMENT:
  • Antepartal history.
  • Emotional status.
  • Vital signs, FHR.
  • Contraction pattern (frequency, duration, intensity).
  • Vaginal discharge.

GOAL = to minimize physical

/ psychological stress during labor/birth. Emotional support.

preterm labor
Preterm Labor:
  • Occurs after 20 weeks gestation and before 38 weeks.
  • Causes may be from maternal, fetal, or placental factors.
  • Prevention:
    • Primary: close observation and eduction in S&S of labor.
    • Secondary: prompt, effective Rx of associated disorders.
    • Tertiary: suppression of preterm labor.
tertiary suppression of preterm labor
Tertiary: suppression of preterm labor
  • Bedrest.
  • Position: side-lying – to promote placental perfusion.
  • Hydration.
  • Pharmacological: betaadrenergic agents to reduce sensitivity of uterine myometrium to oxytocic & prostaglandin stimulation; increase bld flow to uterus.
  • Pt may be maintained at home with adequate follow-up & health teaching.
contraindications for suppression of labor
CONTRAINDICATIONS: for suppression of labor
  • Placenta previa or abruptio placenta.
  • Chorioamnionitis.
  • Erythroblastosis fetalis.
  • Severe preeclampsia.
  • Severe diabetes (brittle).
  • Increasing placental insufficiency.
  • Cervical dilation of 4 cm or more.
  • ROM (depends on cause & if sepsis exists).
nursing assessment ptl
Nursing Assessment: PTL
  • Maternal VS. Response to medication:
    • Hypotension
    • Tachycardia, arrhythmia
    • Dyspnea, chest pain
    • Nausea & vomiting
  • Signs of infection:
    • Increased temperature
    • Tachycardia
    • Diaphoresis
    • Malaise
slide9
Emotional status: denial, guilt, anxiety, exhaustion.
  • Signs of continuing & progressing labor:
    • Effacement
    • Dilation
    • Station

(vaginal exam ONLY if indicated by other signs of continuing labor progress)

  • Status of membranes.
  • FHR, activity (continuous monitoring).
  • Ctx: frequency, duration, strength.
report promptly to md
Report PROMPTLY to MD:
  • Maternal pulse of 110 or more.
  • Diastolic pressure of 60 mmHg or less.
  • Increase in maternal temperature.
  • Respirations of 24 or more; crackles (rales).
  • Complaint of dyspnes.
  • Contractions: increasing frequency, strength, duration, or cessation of ctx.
slide11
Intermittent back and thigh pain.
  • Rupture of membranes.
  • Vaginal bleeding.
  • Fetal distress.

IF LABOR CONTINUES:

  • GOAL = facilitate infant survival; emotional support; support comfort measures; health teaching.
dysfunctional labor pattern
Dysfunctional Labor Pattern
  • Hypertonic labor
  • Hypotonic labor
  • Precipitate labor level

Chapter 26

hypertonic dysfunction
HYPERTONIC DYSFUNCTION:
  • Increased resting tone of uterine myometrium; diminished refractory period; prolonged latent phase.
    • Nullipara: more than 20 hours.
    • Multipara: more than 14 hours.
  • Etiology: unknown. Theory – ectopic initiation of incoordiante uterine ctx.
  • Assessment:
    • Onset (early labor)
slide14
Contractions:
    • Continuous fundal tension, incomplete relaxation.
    • Painful.
    • Ineffectual – no effacement or dilation.
  • Signs of fetal distress:
    • Meconium-stained fluid.
    • FHR irregularities.
  • Maternal VS.
  • Emotional status.
  • Medical evaluation: to rule out CPD.
    • Vaginal examination, x-ray pelvimetry, ultrasonography.
interventions with hypertonic dysfunction
Interventions with Hypertonic Dysfunction:
  • Short-acting barbiturates (to encourage rest, relaxation).
  • IV fluids (to restore / maintain hydration & fluid-electrolyte balance).
  • If CPD – c/s.
  • Provide emotional support.
  • Provide comfort measures.
  • Prevent infection (strict aseptic technique).
  • Prepare patient for c/s if needed.
hypotonic dysfunction
HYPOTONIC DYSFUNCTION:
  • After normal labor at onset, ctx diminish in frequency, duration, & strength.
  • Lowered uterine resting tone; cervical effacement & dilation slow / cease.
  • Etiology:
    • Premature or excessive analgesia / anesthesia (epidural, spinal block).
    • CPD.
    • Overdistention (hydramnios, fetal macrosomia, multifetal pregnancy).
    • Fetal malposition / malpresentation.
    • Maternal fear / anxiety.
slide17
Assessment:
    • Onset (latent phase & most common in active phase).
    • Contractions - normal previously, will demonstrate:
      • Decreased frequency.
      • Shorter duration.
      • Diminished intensity (mild to moderate).
      • Less uncomfortable.
    • Cervical changes – slow or cease.
    • Signs of fetal distress – rare.
      • Usually late in labor d/t infection secondary to prolonged ROM.
      • Tachycardia.
slide18
Maternal VS (elevated temperature) – may indicate infection.
    • Medical diagnosis – procedures: vaginal examination, x-ray pelvimetry, ultrasonography. To rule out CPD (most common cause).
  • Management:
    • Amniotomy (artificial ROM).
    • Oxytocin augmentation of labor.
    • If CPD, prepare for c/s.
    • Emotional support, comfort measures, prevent infection.
precipitate labor
Precipitate Labor
  • Labor that progresses rapidly and ends with the delivery occurring less than 3 hours after the onset of uterine activity.
  • Rapid labor and delivery.
fetal malpresentation and malposition
Fetal Malpresentation and Malposition
  • Breech presentation
  • Shoulder presentation
  • Face presentation
  • Malpositions

Chapter 26

breech presentations
Breech Presentations
  • Fetal descent in which the fetal buttocks, legs, feet, or combination of these parts is found first in the maternal pelvis.
  • Labor tends to be longer and more difficult due to a softer presenting part, that does not fill the birth canal completely.
  • Increase risks for fetal outcome.
shoulder presentation
Shoulder Presentation
  • Fetal descent in which the shoulder precedes the fetal head in the maternal pelvis alone or along with the ftal arm and hand.
  • Vaginally undeliverable.
face presentation
Face Presentation
  • Fetal descent in which hyperextension of the fetal head and neck allows the fetal face to descend into the maternal pelvis, as opposed to flexion that results in fetal vertex presentation.
  • Brow presentation = occurs when the area between the anterior fontanelle and the fetal eyes descends first.
malpositions
Malpositions
  • Persistent occipitoposterior position.
  • Persistent occipitotransverse position.
  • Result from fetal rotation as the fetus descends through the pelvis.
  • Possible precipitating factors are macrosomia and pelvic abnormalities.
  • Results in increased discomfort (particularly back labor), prolonged, abnormal labor, soft tissue injury, lacerations, or an extensive episiotomy incision.
maternal and fetal structural abnormalities
Maternal and Fetal Structural Abnormalities
  • Cephalopelvic disproportion (CPD)
  • Macrosomia

Chapter 26

dystocia
DYSTOCIA:
  • Difficult labor.
  • Causes:
    • “3 Ps” for mother: Psych, Placenta, Position.
    • “3Ps” for fetus: Power, Passageway, Passenger.
  • POWER: forces of labor (uterine contractions, use of abdominal muscles).
    • Premature analgesia / anesthesia.
    • Uterine overdistension (multifetal pregnancy, fetal macrosomia)
    • Uterine myomas.
slide27
PASSAGEWAY: Resistance of cervix, pelvic structures.
    • Rigid cervix.
    • Distended bladder.
    • Distended rectum.
    • Dimensions of the bony pelvis: oelvic contractures.
  • PASSENGER: accommodation of the presenting part to pelvic diameters.
    • Fetal malposition / malpresentation.
    • Fetal anomalies.
    • Fetal size.
hazards with dystocia
Hazards with Dystocia:
  • MATERNAL:

1. Fatigue, exhaustion, dehydration.

2. Lowered pain threshold, loss of control.

3. Intrauterine infection.

    • Uterine rupture.
    • Cervical, vaginal, perineal lacerations.
    • Postpartum hemorrhage.
  • FETAL:
    • Hypoxia, anoxia, demise.
    • Intracranial hemorrhage.
placental abnormalities
Placental Abnormalities
  • Placenta previa
  • Abruptio placentae
  • Other placental abnormalities

Chapter 26

placenta previa
PLACENTA PREVIA
  • Abnormal placement of placenta so that it partially covers the cervix; dilatation results in bleeding, which can be of hemorrhagic proportions.
  • The placenta is located over or very near the internal cervical os.
  • Severe hemorrhage can result from digital palpation of the internal os.
  • Previa is a serious but uncommon complication, occurring in .3-.5% of pregnancies.
slide31
Advanced maternal age and multiparity increase the risk.
  • Painless hemorrhage is symptomatic of previa, often around the end of the 2nd trimester.
  • Clinical diagnosis is reached through ultrasound examination in which the placenta is localized in relationship to the cervix.
  • Manual examination is contraindicated!
  • Management of pregnancy depends on gestational age.
placental abruption
PLACENTAL ABRUPTION
  • Premature separation of the placenta from the uterine wall; usually results in maternal hemorrhage and fetal compromise.
  • Classified as “partial” or “total”.
  • “Total Abruption” – fetal death is inevitable.
  • “Partial Abruption” – the fetus has a chance of survival.
  • Separation of >50% is incompatible with fetal survival.
slide33
Grading of Placental Abruptions:
  • Grade I: Slight vag.bleeding & some uterine irritability. Maternal BP is unaffected & there are normal fibrinogen levels. FHR has a normal pattern.
  • Grade II: External bleeding is mild to moderate. The uterus is irritable. Tetanic ctx may be present. Maternal BP is maintained. FHR shows signs of distress. Maternal fibrinogen level is decreased.
slide34
Grade III: The bleeding may be severe & may be concealed in some instances. Uterine ctx are tetanic and painful. Maternal hypotension may be present. The fibrinogen level is greatly decreased & there are coagulation problems.

Diagnosis: may be made by ultrasound, but frequently the diagnosis is made and confirmed at delivery, by inspection of the placenta.

umbilical cord abnormalities
Umbilical Cord Abnormalities
  • Velamentous insertion of the cord
  • Umbilical cord compression
  • Umbilical cord prolapse

Chapter 26

velamentous insertion of the cord
Velamentous Insertion of the Cord
  • Condition where the umbilical cord joins the placenta at the edge, rather than the typical insertion in the center.
  • Can result in chronic altered fetal perfusion. Can lead to trauma and compression during L&D, resulting in rupture and hemorrhage.
slide37
PROLAPSED UMBILICAL CORD:
  • Cord descent in advance of presenting part; compression interrupts blood flow, exchange of fetal / maternal gases. Leads to fetal hypoxia, anoxia, death (if unrelieved).
  • Etiology:
    • SROM or AROM.
    • Excessive force of escaping fluid (hydramnios).
    • Malposition (breech, compound presentation, transverse lie).
    • Preterm or SGA fetus – allows space for cord descent.
slide38
Assessment:
    • Visualization of cord outside (or inside) vagina.
    • Palpation of pulsating mass on vaginal exam.
    • Fetal distress – variable deceleration and persistent bradycardia.
  • Nursing interventions:
    • Reduce pressure on cord.
    • Increase maternal / fetal oxygenation (O2 per mask @ 8-10 liters).
    • Protect exposed cord (continuous pressure on presenting part to keep pressure off cord).
slide39
Identify fetal response to these measures, reduce threat to fetal survival: moniotr FHR continuously.
  • Expedite termination of threat to fetus (prepare for immediate vaginal or c/s).
  • Support mother and significant other (try to explain things while mobilizing delivery team).
amniotic fluid abnormalities
Amniotic Fluid Abnormalities
  • Polyhydramnios
  • Oligohydramnios
  • Amniotic fluid embolism

Chapter 26

summary of danger signs during labor
Summary of Danger Signs During Labor:
  • Contractions: strong, every 2 min. or less, lasting 90 sec. or more; poor relaxation between ctx.
  • Sudden sharp abdominal pain followed by boardlike abdomen and shock (abruptio placenta or uterine rupture).
  • Marked vaginal bleeding.
  • FHR periodic pattern decelerations – late; variable; absent.
slide42
Baseline FHR:
    • Bradycardia (<100 bpm)
    • Tachycardia (>160 bpm)
  • Amniotic fluid:
    • Amount: excessive; diminished.
    • Odor
    • Color: meconium stained or particulate; port-wine; yellow.
    • 24 hr or more since ROM.
  • Maternal hypotension.
postpartum hemorrhage
Postpartum Hemorrhage:
  • Definition:
    • More than 500cc of blood loss after vaginal birth.
    • More than 1000cc of blood loss after C/S.
  • Blood loss is often underestimated by up to 50% (ACOG, 1998). Subjective.
  • #1 cause of PP Hemorrhage = Uterine Atony.
risk factors for pp hemorrhage
Risk Factors for PP Hemorrhage:
  • Uterine Atony: Marked hypotonia of the uterus
    • Overdistended uterus
    • Anesthesia and analgesia
    • Previous history of uterine atony
    • High parity
    • Prolonged labor, oxytocin-induced labor
    • Trauma during labor and birth
risk factors for pp hemorrhage1
Risk Factors for PP Hemorrhage:
  • Lacerations of the birth canal
  • Retained placental fragments
  • Ruptured uterus
  • Inversion of the uterus
  • Placenta accreta
  • Coagulation disorders
  • Placental abruption
risk factors for pp hemorrhage2
Risk Factors for PP Hemorrhage:
  • Placenta previa
  • Manual removal of a retained placenta
  • Magnesium sulfate administration during labor or postpartum period
  • Endometritis
  • Uterine subinvolution
lacerations
Lacerations:
  • Cervix, vagina, perineum.
  • Suspected when bleeding continues despite a firm, contracted uterine fundus.
  • Characteristics: bleeding can be a slow trickle, an oozing, or frank hemorrhage.
  • Influencing factors: structural, maternal, fetal
  • Lacerations = the most common cause of injuries in the lower portion of the genital tract.
retained placenta
Retained Placenta:

Causes:

  • Partial separation of normal placenta
  • Entrapment of the partially or completely separated placenta by uterine constriction ring
  • Mismanagement of the 3rd stage of labor
  • Abnormal adherence of the entire placenta or a portion of placenta to the uterine wall

Types:

  • Nonadherent retained placenta
  • Adherent retained placenta
inversion of the uterus
Inversion of the Uterus
  • Rare, but life threatening. (1 in 2000-2500 births). May recur with additional births.
  • Contributing factors:
    • Fundal implantation of placenta
    • Vigorous fundal pressure
    • Excessive traction applied to cord
    • Uterine atony
    • Leiomyomas
    • Abnormally adherent placental tissue
uterine subinvolution
Uterine Subinvolution
  • Causes:
    • Retained placental fragments
    • Pelvic infection
  • Signs and symptoms:
    • Prolonged lochial discharge
    • Irregular or excessive bleeding
    • Hemorrhage
    • Pelvic exam reveals a uterus that is larger than normal and may be boggy
assessing cardiac output pph
Assessing Cardiac Output: PPH

NURSING ASSESSMENTS:

  • Palpation of pulses (rate,quality, equality)
  • Auscultation
  • Inspection
  • Observation
meds used to rx pp hemorrhage
Meds used to Rx PP Hemorrhage
  • Oxytocin (Pitocin)
  • Methylergonovine (Methergine; Ergotrate)
  • Prostaglandin F2 (Prostin / 15M; Hemabate)
emergency hemorrhagic shock
Emergency: Hemorrhagic Shock

Assessments:

  • Respirations = rapid and shallow
  • Pulse = rapid, weak, irregular
  • BP = decreasing (late sign)
  • Skin = cool, pale, clammy
  • Urinary Output = decreasing
  • Level of Consciousness = lethargy to coma
  • Mental status = anxiety to coma
  • Central venous pressure = decreased
emergency hemorrhagic shock1
Emergency: Hemorrhagic Shock

Intervention:

  • Summon assistance and equipment
  • Start IV per standing orders (large bore preferable)
  • Ensure patent airway; administer oxygen
  • Continue to monitor status
coagulopathies
Coagulopathies
  • Idiopathic Thrombocytopenic Purpura: (ITP)
  • von Willebrand Disease: a type of hemophilia, factor VIII deficiency, most common congenital clotting defect of women in childbearing years.
  • Disseminated Intravascular Coagulation: (DIC) a pathologic form of clotting, diffuse. Includes platelets, fibrinogen, prothrombin, and factors V and VII.
  • Thromboembolic Disease: formation of clot(s) in blood vessels caused by inflammation or partial obstruction of the vessel.
postpartum infection
Postpartum Infection
  • Antepartal factors:
    • Hx of previous venous trhombosis, UTI, mastitis, pneumonia
    • Diabetes mellitus
    • Alcoholism
    • Drug abuse
    • Immunosuppression
    • Anemia
    • Malnutrition
intrapartal factors
Intrapartal Factors
  • Cesarean birth
  • PROM
  • Chorioamnionitis
  • Prolonged labor
  • Bladder catheterization
  • Internal fetal or uterine pressure monitor
  • Multiple vaginal exams after ROM
intrapartal factors continued
Intrapartal Factors (continued)
  • Epidural anesthesia
  • Retained placental fragments
  • PP hemorrhage
  • Episiotomy or lacerations
  • Hematomas
types of pp infection
Types of PP Infection
  • Endometritis (most common – usually begins as a localized infection at the placental site, but can involve entire endometrium)
  • Wound infections (c/s incision, episiotomy, repaired laceration site)
  • UTIs (2-4 % of PP women)
  • Mastitis (1% of BF moms, usually 1st)
sequelae of childbirth trauma
Sequelae of Childbirth Trauma
  • Uterine Displacement & prolapse
  • Cystocele and Rectocele
  • Urinary Incontinence
  • Genital Fistulas
pp psychologic complications
PP Psychologic Complications
  • Mood Disorders: with or without psychotic features, if the onset occurs within 4 weeks of childbirth.
    • “Baby Blues” – occurs in up to 70% of PP moms
    • Postpartum Depression
    • Postpartum Psychosis
infants with special needs
Infants With Special Needs
  • Priorities
    • Initiation & maintenance of respirations
    • Establishment of extrauterine circulation
    • Control of body temperature
    • Intake of adequate nourishment
    • Establishment of waste elimination
    • Establishment of an infant-parent relationship
    • Prevention of infection
    • Provision of developmental care for mental & social development
high risk infants
High-Risk Infants
  • May need resuscitation at birth.
  • Most institutions require AHA Certification in Neonatal Resuscitation of all personnel at deliveries
  • Requirements may include:
    • Warmth
    • Oxygen
    • Intubation
    • Suctioning
small for gestational age sga
Small for Gestational Age (SGA)
  • Definition: birth weight is below the 10th percentile on an intrauterine growth curve for that age infant.
  • Infant could be preterm, term, or postterm.
  • Have difficulty maintaining body warmth d/t low fat stores; may develop hypoglycemia from low glucose stores.
large for gestational age lga
Large for Gestational Age (LGA)
  • Definition: birthweight is above the 90th percentile on an intrauterine growth chart for that gestational age.
  • Infant could be preterm, term, or postterm.
  • Often are IDM (infants of diabetic mothers), and particularly prone to hypoglycemia or birth trauma.
preterm infants
Preterm Infants
  • Definition: born before 37 weeks of gestation.
  • Particular problems: respiratory function, anemia, jaundice, persistent patent ductus arteriosus, & intracranial hemorrhage.
  • Low-birthweight infants = those weighting 1500-2500 grams.
slide69
Very-low-birthweight infants = those weighing 1000-1500 grams.
  • Extremely-very-low-birthweight infants = those weighing between 500-1000 grams.
  • All such infants need intensive care from the moment of birth.
  • Risks: neurologic after-effects caused by being so critically close to the age of viability.
postterm infants
Postterm Infants
  • Definition: born after 42 weeks gestation.
  • Particular problems: establishing respirations, meconium aspiration, hypoglycemia, temperature regulation, and polycythemia.
respiratory distress syndrome
Respiratory Distress Syndrome
  • Commonly occurs in preterm infants from a deficiency or lack of surfactant in the alveoli.
  • Without surfactant the alveoli collapse on expiration & require extreme force for reinflation.
  • Primary Rx: synthetic surfactant replacement at birth by ET tube insufflation, followed by oxygen and ventilatory support.
transient tachypnea
Transient Tachypnea
  • A temporary condition caused by slow absorption of lung fluid at birth.
  • Close observation of the infant is necessary until the fluid is absorbed and respirations slow to a normal rate.
meconium aspiration syndrome
Meconium Aspiration Syndrome
  • Occurs when infant inhales meconium-stained amniotic fluid during birth.
  • Results in irritation to the airway (from meconium) & may lead to both airway spasm and pneumonia.
  • Infant needs: oxygen, ventilatory support, & possibly antibiotic until the effects of the airway subside.
apnea
Apnea:
  • Definition: a pause in respirations longer than 20 seconds, with accompanying bradycardia.
  • Occurs in preterm infants who have secondary stresses such as: infection, hyperbilirubinemia, hypoglycemia, or hypothermia.
sudden infant death syndrome
Sudden Infant Death Syndrome
  • Definition: the sudden, unexplained death of an infant.
  • Associated with infants sleeping on their stomachs (prone) and infants who were born premature.
  • Nursing prevention: advising parents to position their infant on the back for sleeping.
hyperbilirubinemia
Hyperbilirubinemia
  • Results from: destruction of RBCs, due either to a normal physiologic response or an abnormal destruction of the RBCs.
  • Hemolytic disease of the newborn is destruction of RBCs from Rh or ABO incompatibility.
  • Phototherapy or exchange transfusion is used to prevent kernicterus.
neonatal hemorrhagic disease
Neonatal Hemorrhagic Disease
  • Definition: a lack of clotting ability resulting from a deficiency of vitamin K at birth.
  • Prevention is by injection of vitamin K to all infants at birth.
retinopathy of prematurity
Retinopathy of Prematurity
  • Definition: destruction of the retina due to exposure of immature retinal capillaries to oxygen.
  • Monitoring oxygen saturation via arterial blood gases is an important prevention measure.
infections of the newborn
Infections of the Newborn
  • Streptococcal Group B pneumonia: from maternal GBBS.
  • Hepatitis B infection
  • Ophthalmia neonatorum: from gonococcal and chlamydial conjunctivitis.
  • Herpes Virus infection.
other neonatal risks
Other Neonatal Risks:
  • Infants of diabetic mothers (IDM)
  • Infants of drug abusing women

NOTE: respiratory distress, hypoglycemia, hypo/hyperthermia are common S&S of neonatal infection.