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CORNEALEDEMA

Presentation on corneal edema with references from Khurana, Kanski's, Parsons textbooks

Sudeeptha
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CORNEALEDEMA

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  1. TOPIC: CORNEAL OEDEMA PRESENTED BY: ROLL NOS 66-84 DONE BY: ROLL NO:82 BATCH b

  2. CONTENTS • NORMAL FACTORS • DEFINITION & PATHOPHYSIOLOGY • SYMPTOMS • HISTORY TAKING • SIGNS • CAUSES • TYPES

  3. CORNEAL LAYERS • NORMAL FACTORS • 1) EPITHELIUM • 2) BOWMAN’S MEMBRANE • 3) STROMA • 4) DUA’S LAYER (RESISTANT) • 5) DESCEMET’S MEMBRANE (RESISTANT) • 6) ENDOTHELIUM • FACTORS WHICH DRAW WATER INTO CORNEA • 1)INTRAOCULAR PRESSURE (IOP) • 2) SWELLING PRESSURE OF STROMA- This is the tendency of stroma to swell due to interfibrillary proteoglycans and other proteins. • FACTORS WHICH DRAW WATER OUT • 1)ACTIVE PUMPING ACTION OF CORNEAL ENDOTHELIUM 2)MECHANICAL BARRIER ACTION OF ENDOTHELIUM AND EPITHELIUM

  4. DEFINITION • COMMON CAUSE OF LOSS OF CORNEAL TRANSPARENCY • ACCUMULATION OF FLUID IN THE LAYERS OF CORNEA • - ESPECIALLY BETWEEN BASAL CELLS (columnar cells of epithelium) • -BETWEEN LAMELLAE • -AROUND NERVE FIBRES OF STROMA (STROMAL HAZE)

  5. IMBIBITIONAL PRESSURE= IOP - SP

  6. SYMPTOMS • Maybe ASYMPTOMATIC • REDUCED VISION- DUE TO STROMAL HAZE • PAIN • PHOTOPHOBIA • LACRIMATION, REDNESS • COLOURED HALOS (*note- Fincham’s stenopaic slit test= remains intact) • FOREIGN BODY SENSATION

  7. HISTORY TAKING • AGE OF ONSET • DURATION • LATERALITY • FAMILY HISTORY • OCULAR MEDICATIONS • H/O SURGERY/TRAUMA • DIURNAL VARIATION

  8. SIGNS STROMA EPITHELIAL • DUE TO EPITHELIAL DEFECTS (fluorescein stain) • LUSTER LOST • MICROCYST • BULLOUS KERATOPATHY (mostly pain) • SLIT WIDER THAN NORMAL ON SLIT LAMP • DESCEMET MEMBRANE FOLDS (mostly reduced vision)

  9. BULLOUS KERATOPATHY MICROCYST STAGE PERIPHERAL EDEMA (Brown Mclean syndrome) DESCEMET MEMBRANE FOLDS BREAKS IN DESCEMET’S (Forceps injury)

  10. ENDOTHELIAL EXAM • GUTTAE SEEN • THESE ARE DROP LIKE ACCUMULATIONS ON POSTERIOR SURFACE OF CORNEA • GIVES DIMPLES/BEATEN METAL APPEARANCE ON SLIT LAMP • IN CASES LIKE FUCHS’ DYSTROPHY

  11. CAUSES • Mostly acute and stromal edema seen • INCREASED INTRAOCULAR PRESSURE (normal endothelium, acute angle closure glaucoma cases) • MECHANICAL INJURIES • CHEMICAL BURNS • RADIATIONAL INJURIES • THERMAL INJURIES • INFLAMMATION AND INFECTIONS (Herpes) INJURIES- BIRTH TRAUMA (Forceps delivery),SURGICAL TRAUMA DRUGS/ HYPOXIA (acute and stromal edema) ENDOTHELIAL FAILURE- Fuchs’ Dystrophy, Congenital Hereditary endothelial dystrophy, Posterior polymorphous dystrophy, Iridocorneal endothelial syndrome (chronic, progressive stromal edema) INFLAMMATIONS- Uveitis, Endophthalmitis, Corneal graft infection

  12. EDEMA IN AN INFANT DUE TO FORCEPS DELIVERY FUCHS’ DYSTROPHY GRAFT REJECTION EDEMA EDEMA IN PHACOMORPHIC GLAUCOMA HERPES INFECTION EDEMA

  13. TYPES • GROUND GLASS APPEARANCE • MINIMAL CHANGE • PRESENTS WITH CONTRAST SENSITIVITY • MICROCYSTIC APPEARANCE STROMAL AND EPITHELIAL EDEMA (FS)

  14. TREATMENT • TREATING ASSOCIATED CAUSES • INCREASED IOP- carbonic anhydrase inhibitors • INFLAMMATION- STEROIDS • TREATING EDEMA • HYPERTONIC AGENTS- 5% SODIUM CHLORIDE DROPS OR ANHYDROUS GLYCERINE • HOT FORCED AIR FROM HAIR DRYER • THERAPEUTIC SOFT CONTACT LENS • ENDOTHELIAL KERATOPLASTY • PENETRATING KERATOPLASTY- FOR LONG STANDING CASES

  15. THANK YOU • BIBLIOGRAPHY • A K KHURANA • PARSONS’ DISEASES OF THE EYE • KANSKI’S CLINICAL OPHTHALMOLOGY • OPHTHALMOLOGY ATLAS BY WILLS EYE INSTITUTE

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