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Pathophysiology. Chronic AR: LV overload ? LV dilatation, eccentric hypertrophy, insidious prog to CHF (decades, typically)Acute AR: nl LV poorly tolerates sudden increase LVEDV ? massive increase LVEDP leading to pulm edema, hypotension /- cardiogenic shock. Epidemiology. Overall prevalence 4.9%; moderate or greater severity 0.5% (Framingham)No sex or racial predilectionAge: dependent on etiology.

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