Mentoring the Mentor

1. Mentoring the Mentor Stuart White, DC, DACBN, CCN Whole Health Associates 1406 Vermont Houston, Texas 77006 713/522-6336 stuartwhite@wholehealthassoc.com www.wholehealthassoc.com www.doctorofthefuture.org

2. Mentor goals: • To declare what is possible and establish a commitment to that possibility • Address personal and professional barriers limiting the ability to serve • Evolution of vision/mission/ethics that drive success • Create immediate action steps to apply learning and growth • Construct the round table of applied trophologists

3. Mentoring the mentor: • Who are the mentors? – Practitioners • Who are we mentoring? – Patients and GAP • What’s the purpose? – Optimized life • How does it work? – Whatever you learn you teach someone else (anyone else) • Who’s is included? – Self selection, you pick yourself

4. Mentoring the mentor: • Each participant attends monthly teleconferences (1 hour in duration, 4th Thursday of month) creating a round table discussion/exploration of the dynamics and details of a nutrition-based wholistic practice • Each participant chooses a colleague in his/her world to convey the notes and information – no information squandering • Issues/problems/questions are considered a learning process for everyone, although individual’s remain anonymous • All questions, comments, case studies to be directed through email to SP rep who will compile and include in next teleconference ( must be submitted 10 days prior)

5. Approach to wisdom Tell me What is it that you plan to do With your wild and precious life? Mary Oliver

6. #2 Core Physiologic Principal Glycemic balance Introduction glucose – source in starch, alcohol, sugars Increased blood glucose Hunger – Sugar cravings Insulin up-regulation Adaptation – Insulin Resistance Cells absorb sugar – store as triglycerides Anabolic weight/fat gain Protein/fat Increased satiety Hypoglycemia – blood sugar too low Stress – Increased Cortisol Glycogen reserve depletion Increase gluconeogenesis Increase inflammation & anxiety Glycemic dysregulation Adrenal /Pancreas stress food dependency / fatigue Mitochondria proliferate Increased energy/stamina/productivity/independence

7. Cortisol vs. Insulin • Fasting glucose will often reveal mild elevation of the glucose bringing to mind dietary glycemic load as a cause • It is also possible for cortisol dysregulation to elevate fasting glucose by promoting glucose liberation from glycogen stores • As clinicians it is useful to distinguish between glucose sourced from ingested glycemic load and elevated cortisol for a variety of reasons • The clinical key to this insight is the triglycerides

8. Clinical lab values • If the fasting glucose is over 100 and it corresponds to triglycerides over 80 it implies that the dietary contribution of glucose exceeds a modest insulin metabolism and thus triglycerides are being generated • If fasting glucose exceeds 100 and the triglycerides are under 80 this implies the dietary glycemic load is not requiring insulin in excess and that the sugar elevation is secondary to the action of cortisol • The following sample blood laboratory values reveal this pattern and help aim the accuracy of the clinical intervention

14. Uric Acid and Fructose Burden • In the past we have reviewed that new thinking around overall fructose burden revealing itself in uric acid levels over 5.5 • This level is still well within normal range but it reveals a burden of the inflammatory pathways resulting in uric acid production • Reducing the daily fructose consumption t6o less than 25 grams can result in achieving normal triglycerides and uric acid less than 5.5

15. 3 targets for waist thickening • Waist thickening plaguing both the overweight and underweight patient is usually an indication of elevated cortisol influences • There are three clinical ways to change the shape of things: 1 – Reduce dietary glycemic load until triglycerides reduce to under 80 indicating a minimal insulin lifestyle 2 – Reduce fructose burden as a next step until the uric acid reduces to under 5.5 and the triglycerides fall to less than 80 3 – Reduce cortisol activity by preventing hypoglycemic events and thus stopping glucose saturation from glycogen reserves

18. What does it mean clinically? • If a person is plateaued in their evolution toward lean body state we must consider fructose overload • This can be done by comparing and contrasting the triglycerides and uric acid to infer the ingestion and metabolic load of too much fructose • This also takes into account reactive hypoglycemia trends in some more severe than others • We begin by keeping the fructose daily consumption below 25 grams • One can of soda will exceed the daily limit

19. Fructose burden – 15-25 grams • It is possible that this is the single factor in the SAD that is robbing Americans of their birthright to health and happiness • As clinicians it is time for us to take our patients as far as they wish to find their perfect health – we can asses this and move people beyond that phase II shift all the way to remarkable states of physiology • 2 out of 3 are overweight, 1 out of 3 are obese – it has become clear that fructose is the single most important factor in this epidemic

20. Assessing Fructose Burden -Fruit Fructose Content Seek to limit daily consumption of fructose to 25 grams per day to avoid fatty degeneration

21. Reduce cortisol • Cortisol is most influenced by low blood sugar levels requiring cortisol elevation to release sugar • The best way to reduce cortisol responses is by eating six meals a day of low glycemic foods resulting in lower cortisol levels • If cortisol stays elevated and a low glycemic diet is adopted ultimately the waist will stay thick – this can lead unwittingly to immune depression and crisis • Watch the waist thickness as best indicator of cortisol function

22. Approach to wisdom When we tug at any one thing in nature We discover it is attached to Everything else J. Muir

23. Change the world It wants to