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Physiological basis of the care of the elderly client

Physiological basis of the care of the elderly client. Cardiovascular System. Patient scenario. RB, 73 year old Caucasian male Medical diagnosis hypertension Prescribed Norvasc, 5 mg qd and Accupril 10 mg BID Often forgets his evening dose Wants “one pill once-a-day”

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Physiological basis of the care of the elderly client

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  1. Physiological basis of the care of the elderly client Cardiovascular System

  2. Patient scenario • RB, 73 year old Caucasian male • Medical diagnosis hypertension • Prescribed Norvasc, 5 mg qd and Accupril 10 mg BID • Often forgets his evening dose • Wants “one pill once-a-day” • Complains of frequent headache on waking • …pill makes him urinate too much • …he has a cough that won’t go away • …feels fine, maybe he doesn’t need it after all

  3. Informal evaluation What additional information do you need? • Subjective information • Objective information • Psychosocial information

  4. The cardiovascular system • Supplies oxygen to all parts of the body • A failure in this system creates a cascade of failure in other systems • Regardless of nursing focus, you are likely to encounter cardiovascular concerns in the elderly client due to normal age related changes

  5. Review of cardiac structure and function • Circulation is established by electrical system of the heart • Left side of heart produced enough force to overcome systemic resistance • Effective circulation due in part to one way valves between the chambers of the heart • Effective circulation is also dependent upon sequential contraction and relaxation of the heart

  6. Potential challenges for the elderly client

  7. Preload and afterload • Preload is a representation of the pressure stretching the left ventricle after passive filling and atrial contraction (diastole) by the blood returning to the heart • Afterload is the amount of pressure produced by the left ventricle in order to contract (systole)

  8. Regulation of cardiac function

  9. Left ventricle efficiency • Determined by amount of blood pumped from the left ventricle at end of diastole • Affected by • Strength of contraction • Amount of blood in the ventricle • Competency of the valves • Peripheral vascular resistance • Ejection fraction = stroke volume / left ventricle end diastolic volume

  10. The electrocardiogram • Normal PR = .12-.20 (3-5 □s) • Normal QRS = < .12 (3 □s) • Normal sinus rhythm • Each P wave followed by QRS • Rate 60-90 with <10% variation • P wave: atrial depolarization • QRS complex: ventricular depolarization • T wave: ventricular repolarization • Little boxes = .04 sec; Big boxes = 5x.04 = .2 sec

  11. Normal age related changes • Heart valves become stiff • Decreased renin, angiotensin and aldosterone production • Arterial stiffening and loss of elasticity • Veins thicken and valvular reflux occurs • Decreased baroreceptor sensitivity • Decrease in number of normal pacemaker cells in sinus node • Myocardial hypertrophy • Increased size of myocardial cells • Thickening of left ventricular wall

  12. Resulting systemic effects • Increased resistance of peripheral vessels • Decreased coronary blood flow • Reduced cardiac output • Less efficient cardiac oxygen usage • Slower response to cardiac challenge if not in good physical condition

  13. To prevent debilitation from cardiovascular changes through lifestyle modification… • Remain physically active—30 minutes aerobic activity per day most days of the week • Avoid obesity—maintain normal BMI between 22-25 • Avoid smoking • Control blood pressure • Control cholesterol levels • Restrict sodium intake to 2.4 g/day • Limit alcohol to ≤2/day for men, ≤1/day for women

  14. Atypical presentation of cardiac disease • Presenting complaint may be heartburn, nausea, fatigue • Mental status changes • Dizziness and falls • Agitation • Sudden change in cognitive abilities • New onset atrial fibrillation • Particularly in women: • Fatigue • Sleep disturbances • Epigastric pain

  15. Heart disease in elderly women Symptoms may be unrecognized: • Sleep disturbances • Intermittent chest tightness, squeezing, pressure • Back, neck, stomach, jaw discomfort • Shortness of breath, nausea, lightheadedness • Break out in cold sweat

  16. Hypertension • A major risk factor for developing other cardiovascular conditions because: • It does not always produce its own symptoms • Many are unaware they have hypertension • It is easily ignored

  17. Classifications of blood pressure

  18. JNC 7 (2003) Guidelines(JNC 8 in development) • After age 50, SBP >140 is a more important risk factor than DBP • A 90% risk of developing HTN exists even in those age 55 who are normotensive • 120-139/80-89 is prehypertensive; patients should begin lifestyle modifications • Most patients with HTN need 2+ medications • Thiazide diuretics should be used to treat uncomplicated HTN • Effective therapy requires patient motivation • Empathy builds trust and promotes motivation

  19. “Instant” teaching points regarding HTN • It is not the same as anxiety • Once you are diagnosed, you are on medication for life* • It is defined as systolic blood pressure > 140 mmHg • Most cases of HTN are classified as primary HTN—the underlying cause is not known *some exceptions!

  20. Results of untreated hypertension • Atherosclerosis of the aorta and large vessels accelerates • Left ventricular hypertrophy develops • Proteinuria due to increased renal arteriole pressure • Vascular changes in the retina (A-V “nicking”) • Increased stroke risk

  21. Nursing management—patients with HTN • Evaluate BP bilaterally and in lying, sitting and standing positions • Blood pressure varies with time of day and with activity • Respond to “white coat hypertension” • Home blood pressure monitoring must be confirmed • Assess for target organ damage with each encounter

  22. Nursing management—patients with HTN • High blood pressure screening • Promote healthy lifestyles • Low fat diet • Low sodium diets • Weight control • Exercise • Smoking cessation • Controlled alcohol consumption • Monitor effects of medication

  23. Medication management of hypertension • Initial treatment usually involves diuretics • Second medication selected pertaining to patient’s health status • β-blockers can cause bradycardia, fatigue, exercise intolerance • Postural hypotension can occur with adrenergic inhibitors and α-blockers • Dry cough, hyperkalemia can occur with ACE inhibitors and angiotensin receptor blockers • Benzothiazepines may cause decreased cardiac output and slow conduction

  24. Hypotension • Frequently associated with medication side effects • Decreased responsiveness of sympathetic nervous system with age affects autoregulation of cardiac output • Lying/sitting (postural) blood pressure: • Supine for at least 5 minutes, then check blood pressure • Check again after 1 and 3 minutes of sitting or standing

  25. Hyperlipidemia • Elevated cholesterol is a risk factor for cardiovascular disorders • Remember…Keep HDLs high, keep LDLs low! • LDL < 100 mg/dl* • HDL > 60 mg/dl* *Optimal per JNC7!

  26. Benefits of the “statins” • Lower LDL cholesterol • Anti-inflammatory • Antithrombotic • Protect plaque stability • Generally well tolerated • Atorvastatin (Lipitor) • Fluvastatin (Lescol XL) • Lovastatin (Mevacor) • Pravastatin (Pravachol) • Rosuvastatin (Crestor) • Simvastatin (Zocor)

  27. Metabolic syndrome (“syndrome X”)

  28. Characteristics of metabolic syndrome • Abdominal fat cells secrete hormones promoting heart disease and diabetes • Patients have below-normal HDL • Decreased insulin sensitivity (level of insulin required to process glucose)

  29. Treatment plan for metabolic syndrome • Cholesterol lowering drugs • Antihypertensives • Diet high in omega-3 fatty acids • Avoid processed foods • Exercise 30-45 minutes moderate intensity

  30. Ischemic heart disease in the elderly • Chest pain is not always present • Fatigue • Weakness • Shortness of breath • GI disturbances

  31. Chest pain • Caused by a mismatch between what the body is able to deliver and what the body requires • Supply ischemia—due to decreased blood flow to the heart • Demand ischemia—due to increased demand for oxygen • In stable angina, chest pain is relieved with rest • If not relieved by rest, can progress to myocardial infarction

  32. Other causes of chest pain • Pericarditis • Heartburn, ulcers • Chondritis • Pulmonary embolus, pneumonia • Herpes zoster

  33. Treatment of angina • Nitroglycerine—vasodilator • Treatment of choice • Comes in tablets, sprays, patches, ointment, IV, sublingual • Tablets for acute attacks • Transdermal, capsules, ointments do not work rapidly enough during acute attacks • Repeat tablet every 5 minutes for acute attack • If no resolution after 3 tablets, patient must be transported to hospital

  34. Myocardial infarction findings • Occurs in stages, treatment directed to the stage • EKG changes—ST elevation • Q wave represents infarcted tissue • CK-MB elevation 4 to 6 hours after infarction • Troponin elevation 6 to 8 hours after infarction • Hemodynamic monitoring necessary if heart failure suspected

  35. Complications of MI • Arrhythmia (dysrhythmia) • Conduction blockages • Heart failure • Pulmonary edema • Ventricular aneurysm • Pericarditis

  36. Anticoagulation treatment of MI • Useful within first few hours of event • Chew an aspirin while waiting for ambulance! • Not all patients are candidates for thrombolytic therapy

  37. Aortic stenosis • Most common valvular disorder in the elderly • Usually due to calcification • Risk factors: • Hyperlipidemia • Diabetes • Hypertension • Left ventricular hypertrophy • Heart failure

  38. Heart failure • Heart no longer able to provide sufficient cardiac output • Men develop after an MI; women after long-standing HTN • Compensatory events • Increased heart rate • Renin → angiotensin I → angiotensin II → increased BP and sodium and water retention • Risk factors: • Coronary artery disease • Hypertension

  39. Right sided versus left sided failure

  40. Neck vein distention

  41. Dysrhythmias (not “arrhythmias!”) • Atrial fibrillation most common dysrhythmia • Incidence increases with age • Not life-threatening by itself; can increase mortality • No P-wave • Disorganized electrical impulses overwhelm SA node • Results in an irregular heart rhythm • Treated with anticoagulation (Heparin, Warfarin [Coumadin])

  42. Venous disease • Valvular incompetence • Pressure transferred to capillaries of lower extremities • Cells break down • Debris collects • Can cause nonhealing ulcers • Often misinterpreted as “spider bite” • Treatment is compression

  43. Formal evaluation • What is your nursing diagnosis for RB? • What is your desired outcome? • What are appropriate interventions pertinent to your desired outcome?

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