slide1
Download
Skip this Video
Download Presentation
به نام خداوند جان و خرد

Loading in 2 Seconds...

play fullscreen
1 / 13

به نام خداوند جان و خرد - PowerPoint PPT Presentation


  • 145 Views
  • Uploaded on

به نام خداوند جان و خرد . Otosclerosis. Back ground: METABOLIC BONE DISEASE IN OTIC CAPSULE. ETIOLOGY. 1-GENETICALLY MEDIATED AUTOSOMAL DOMINANT 2-VIRUSES: MEASLES SYMOTOMS: CHL – SNHL – MHL AGE: 10 – 45 years RACE – 10 % WHITE POPULATIONS 0.5% ASIANS 0.1% AFRICAN 75 – 80% BILATERAL.

loader
I am the owner, or an agent authorized to act on behalf of the owner, of the copyrighted work described.
capcha
Download Presentation

PowerPoint Slideshow about ' به نام خداوند جان و خرد ' - taline


An Image/Link below is provided (as is) to download presentation

Download Policy: Content on the Website is provided to you AS IS for your information and personal use and may not be sold / licensed / shared on other websites without getting consent from its author.While downloading, if for some reason you are not able to download a presentation, the publisher may have deleted the file from their server.


- - - - - - - - - - - - - - - - - - - - - - - - - - E N D - - - - - - - - - - - - - - - - - - - - - - - - - -
Presentation Transcript
slide2

Otosclerosis

  • Back ground:
  • METABOLIC BONE DISEASE IN OTIC CAPSULE
etiology
ETIOLOGY

1-GENETICALLY MEDIATED AUTOSOMAL DOMINANT

2-VIRUSES: MEASLES

SYMOTOMS: CHL – SNHL – MHL

AGE: 10 – 45 years

RACE – 10 % WHITE POPULATIONS

0.5% ASIANS

0.1% AFRICAN

75 – 80% BILATERAL

pathophysiology
PATHOPHYSIOLOGY

1- EARLY OTOSPONGIOTIC PHASE

2- LATER OTOSCLEROTIC PHASE

epidemiology
EPIDEMIOLOGY

FREQUECY: HISTOLOGICALLY OTOSCLEROSIS: 10%

CLINICALLY OTOSCLEROSIS: 1%

MORTALITY & MORBIDITY:

1- CHL

2- SNHL

3- MHL

4-TINNITUS

5- VERTIGO

slide6

RACE: MORE COMMON IN WHITE PERSONS

SEX: FEMALE -2

MALE -1

AGE: CLINICAL OTOSC. CAN MANFEST AS AGE 7 -8 YEARS

MOST COMMONLY IN AGE 15 – 35 YEARS

PICK INCIDECE: 3TH DECADE

symptoms
SYMPTOMS

1- HEARING LOSS: CHL – SNHL – MHL

2- TINNITUS

3- DIZZINESS

4- ROTATORY VERTIGO

physical exams
PHYSICAL EXAMS

1- PUR TONE AUDIOMETRY

2- TYMPANOMETRY

3- TUNING FORKS TESTS: RINNE TEST WEBER TEST

4- SCHWARTZE SIGN

diferential diagnosis
DIFERENTIAL DIAGNOSIS

1- OTITIS MEDIA

2- CONGENTIAL STAPES FIXATIONS

3- GONGENITAL MALLEAL HEAD FIXATION

4- TYMPANOSCLEROSIS

5- PAGET DISEASE

6- OSTEOGENESIS IMPERFECTA

imaging studies
IMAGING STUDIES

1- CTSCAN: HALO SIGN

  • OTHER TESTS
  • DIAGNOSIS: COMBINATION OF
  • 1- ADIOMETRIC TESTS:
  • A- CARHART NOTCH
  • B- SDS
  • C- TYMPANOGRAM
  • D- ABSENT STAPEDIUS REFLEX

2- HISTORICAL FEATURES

A- FINE – CUT CT SCANNING

B- VESTIBULAR TEST, NG

treatment
TREATMENT

1- MEDICAL CARE

A- SODIUM FLUORIDE 20 – 120 mg/D

B- CALCIUM CARBONATE

C- VITAMIN D

EFFECTIVENESS:

A- DISAPPEARANCE OF SCHWARTZE SIGN

B.AUDIOMETRIC TESTS

C. FOLLOW UP CT SCANNING

2-HEARING AIDS

3-SURGICAL CARE

A- GENERAL ANEST.

B-LOCAL ANEST.

1-STAPEDECTOMY

2-STAPEDETOMY

complications
COMPLICATIONS

SNHL 1 – 2%

PERMANENT FACIAL N. INJURY PROBABLY <1 PER 1000 CASES

TYMPANIC MEMBRANE PERFORATIONS 1 – 2%

ALTERATION OF TASTE

DYSEQUILIBRIUM AND VERTIGO WITH NAUSEA AND VOMITING.

TINNITUS

prognosis
PROGNOSIS

CHL 50 – 60 dB

SNHL TOTAL DEAFNESS

ad