training adaptations
Download
Skip this Video
Download Presentation
Training Adaptations

Loading in 2 Seconds...

play fullscreen
1 / 20

Training Adaptations - PowerPoint PPT Presentation


  • 83 Views
  • Uploaded on

Training Adaptations. LIVER. SKELETAL MUSCLE TISSUE. Adrenal Gland. Adipocytes. Mitochondria. TRAINING WILL: Decrease RER Does not effect sub-max oxygen uptake Increases LT and lowers muscle and blood lactate at any sub-max workload.

loader
I am the owner, or an agent authorized to act on behalf of the owner, of the copyrighted work described.
capcha
Download Presentation

PowerPoint Slideshow about ' Training Adaptations' - sherry


An Image/Link below is provided (as is) to download presentation

Download Policy: Content on the Website is provided to you AS IS for your information and personal use and may not be sold / licensed / shared on other websites without getting consent from its author.While downloading, if for some reason you are not able to download a presentation, the publisher may have deleted the file from their server.


- - - - - - - - - - - - - - - - - - - - - - - - - - E N D - - - - - - - - - - - - - - - - - - - - - - - - - -
Presentation Transcript
slide2

LIVER

SKELETAL MUSCLE TISSUE

Adrenal Gland

Adipocytes

Mitochondria

slide3

TRAINING WILL:

Decrease RER

Does not effect sub-max oxygen uptake

Increases LT and lowers muscle and blood lactate at any sub-max workload

slide4

However, when express per gram of mitochondrial protein training does not alter specific activity.

Aerobic training can cause 50-100% increases in mitochondrial mass per gram of skeletal muscle.

slide8

Total CHO:

Pre = 145 umol/min

Post = 100 umol/min

or a 50% decrease

This is due to decrease reliance on blood glucose and muscle glycogen

slide9

Training decreases Ra(rate of appearance) of glucose from the liver. This means less glycogen depletion in liver.

slide11

With training you can double your mitochondrial mass, thus at any giving work load each mitochondrion will only be working (ie producing ATP) at half the rate it was before training. The main stimulus for increases in oxidative phosphorylation is ADP. Therefore the increase in intracellular ADP must be less in trained individuals.

training adaptations1
Training Adaptations

 # mitochondria

  •  blood lactic acid

 epi/norepi release during exercise

 muscle & liver glycogen use

 intramuscular fat use

 # MCT’s (via  mitochondria)

 CAT I (ß-oxidation)

 cAMP

training adaptations cont
Training Adaptations (cont.)
  • Two major changes that occur with training
  • 1.)  # mitochondria in muscle cells
    • can be doubled at most
  • 2.)  epi/norepi release during exercise
    • training  sympathetic activity at any given work load
benefits of adaptations to training
Benefits of adaptations to training
  • 1.)  Glycolysis
    • Spares CHO, liver glycogen
    • maintains blood glucose (CNS)
    • mechanisms:
      •  catecholamines   PFK activity
      •  mitochondria  faster ATP generation,  [AMP&ADP]   PFK activity
  • 2.)  Blood Lactic Acid
    • keeps blood pH normal
    • mechanisms:
      •  glycolysis
      •  mitochondria (more MCTs)
benefits of adaptations to training cont
Benefits of adaptations to training (cont.)
  • 3.)  Fat use
    •  fat use from adipocytes via  epi/norepi
    •  fat use from intramuscular TGs
    • mechanisms:
      •  mitochondria (via  in CAT I activity)
      •  acetyl CoA production   PFK   glycolysis
  • Take home point:
    • *Training  glycolysis &  Fat use via  mitochondria &  catecholamines*
ad