'Rb gene' presentation slideshows

Neoplasia Lecture 3

Neoplasia Lecture 3. CARCINOGENESIS. Dr. Maha Arafah Dr. Abdulmalik Alsheikh , MD, FRCPC. Foundation block 2012 Pathology. CARCINOGENESIS. Carcinogenesis is a multistep process at both the phenotypic and the genetic levels. It starts with a genetic damage: Environmental Chemical

1.04k views • 98 slides

914 views • 77 slides

Neoplasia Lecture 3

Neoplasia Lecture 3. CARCINOGENESIS. Dr. Maha Arafah Dr. Abdulmalik Alsheikh , MD, FRCPC. Foundation block 2012 Pathology. CARCINOGENESIS. Carcinogenesis is a multistep process at both the phenotypic and the genetic levels. It starts with a genetic damage: Environmental Chemical

1.22k views • 71 slides

Neoplasia Lecture 3

Neoplasia Lecture 3. CARCINOGENESIS. Dr. Maha Arafah Dr. Abdulmalik Alsheikh , MD, FRCPC. Foundation block 2012 Pathology. CARCINOGENESIS. Carcinogenesis is a multistep process at both the phenotypic and the genetic levels. It starts with a genetic damage: Environmental Chemical

903 views • 68 slides

CHAPTER 16

CHAPTER 16. Cancer. Introduction. Cancer results from alterations in the DNA of a somatic cell during the lifetime of the affected individual. Gene expression in cancer cells is altered. Cancer cells proliferate in an uncontrolled manner, forming malignant tumors.

777 views • 63 slides

CHAPTER 16

CHAPTER 16. Cancer. Introduction. Cancer results from alterations in the DNA of a somatic cell during the lifetime of the affected individual. Gene expression in cancer cells is altered. Cancer cells proliferate in an uncontrolled manner, forming malignant tumors.

632 views • 63 slides

Pediatric tumors

Pediatric tumors. Dr Ramadas Nayak Professor & HOD Pathology Yenepoya Medical College Mangalore. Tumors and Tumor-like Lesions of Infancy and Childhood. Only 2% of all malignant tumors occur in infancy and childhood. Benign tumors are even more common than cancers.

549 views • 53 slides

Lecture 8

Lecture 8. Oncogene and anti-oncogene. Zhihong Li ?????. Normal cells. Benign. Malignant cancer. Cancer cells vs. normal cells: what's different?. Cells normally grow, reproduce and die in response to signals inside and outside the body in an orderly way.

1.05k views • 46 slides

Neoplasia 6

Neoplasia 6. Dr Heym Awad FRCPath. Cyclins and cyclin dependent kinases. Normal cell cycle. All the stimuli mentioned in the previous lecture aim for quiescent cells to enter the cell cycle

413 views • 41 slides

The story of Retinoblastoma

Tumor Suppressor Genes. The story of Retinoblastoma. Retinoblastoma is a cancerous disease. 1/20,000 children; 300 per year Average age is 18 months Treatment: enucleation = eye removal Prognosis is good after enucleation over 90% survival with early detection and treatment.

952 views • 40 slides

Neoplasms of infancy and childhood

Neoplasms of infancy and childhood. Neoplasms of infancy and childhood. Benign>malignant Incidence of malignancy:1-15 yrs - 1.3 /10,000 /year but leading cause of death after accidents in the West. (developing countries??)

510 views • 29 slides

Tumor Suppressor Genes p53 &PTEN

Tumor Suppressor Genes p53 &PTEN. G.Mathan Ph.D Assistant Professor. Characteristics of Cancer. Activated proto-oncogene. Inactivated tumour suppressor gene. For cancer: two classes of cellular genes are targets for mutations PROTO-ONCOGENES TUMOUR SUPRESSOR GENES.

298 views • 28 slides

Introduction to Oncology

Introduction to Oncology. Dr. Saleh Unit 9. R.E.B, 4MedStudents.com 2003. Retroviruses. Retroviruses are members of one family of RNA viruses that cause cancer in variety of animals and humans.

370 views • 22 slides

Hallmarks of Cancer Six fundamental changes

Hallmarks of Cancer Six fundamental changes. Self sufficiency in growth factors Insensitivity to growth-inhibitory signals Evasion of apoptosis Limitless replicative potential Sustained angiogenesis Ability to invade and metastasize. Tumor Suppressor Genes.

403 views • 14 slides