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Dentin Hypersensitivity

Dentin Hypersensitivity. Dentin H ypersensitivity. Dentin hypersensitivity is sensation felt, defined as a sharp short pain arising from exposed dentine Sensitivity is caused by open dentinal tubules in response to stimuli Common dental painful disease

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Dentin Hypersensitivity

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  1. Dentin Hypersensitivity

  2. Dentin Hypersensitivity • Dentin hypersensitivity is sensation felt, definedas a sharpshortpainarisingfromexposeddentine • Sensitivity is causedbyopendentinaltubulesinresponsetostimuli • Commondentalpainfuldisease • Oftenunderreportedbypatients

  3. Prevalence of dentin hypersensitivity • globalprevalenceintheadultdentatepoulation 57% • Patientswithperiodontitisincidenceextremehighas 60 to 98% • Symptomscanpresentatanyagefrom 15 years old totheages more tnan 70 • Ages 20 to 40 is a peakprevalence • Womenare more frequentlyaffectedthanmales

  4. Stimuli • Thermal (mainlycold) • Evaporate • Tactile (probe, toothbrush) • Osmotic (sweet, sourfoods) • Chemical

  5. Stimulus • Cold 80% • Air 23% • Touch 20% • Heat 13% • Dietary acid 6.9% • Endogenosus acid 0.7%

  6. Epidemiology • Affected teeth • Premolars 38% • Incisors 26% • Canines 25% • Molars 12% • Location • Cervical 90% • Buccal, occlusal surfaces more prevalent

  7. Structure of dentin • Largestportion of thetoothstructure • Inorganicmaterial (hydroxyapatite) account for 70-75% byweight • 18-20% organicmaterial • 5% water and othermaterials • Dentin is packedwithdentinaltubulesthattransverseitsentirethickness

  8. Dentin tubules • Tubulesareconical, being wideratpulpal end thantheperiphery • Tubulesrunparalelltoeachotherin an S shapecourse • Diameter of tubules: 1-5 Micrometerwith an average of 1,5 • Number of tubulesdecreasesfromabout 65000/mm2 closetothepulptoabout 15000/mm2 nearthe DEJ • Contenttubular fluid • Constantpulpalpressure 25-30 mmHg • Externalstimuliwillcausemovement of thedentinal fluid, a hydrodynamicmovement

  9. Structure of dentin

  10. Dentin hypersensitivitymechanismstheory • Neuraltheory • Odontoblastictransductiontheory • Odontoblastmovementtheory • Hydrodynamictheory (the most widelyacceptedmechanism)

  11. Mechanism of Hypersensitivity The Hydrodynamic Theory: • Exposed dentin tubules • Stimulus (thermal, mechanical, chemical) • Change in rate of dentin fluid flow • Generation action potentials in intradental nerves • Movement of fluid stimulate the free nerve endings (of the A-beta, A-delta) located around the odontoblast body • Action potentials pass to brain to cause pain

  12. Aetiology Two phases development: • Lesion localisation- exposure of dentin • Lesion initiation- opening of dentin tubules (removal of cementum or smear layer)

  13. Aetiology • Loss of enamel – tooth wear • Gingival recession

  14. Loss of enamel • Abrasion • Erosion • Attrition • Abfraction • Fracture

  15. Abrasion • Resultingdirectfrictionforcesbetweentheteeth and theexternalobjects • Toothbrushabrasion is the most common

  16. Abrasion

  17. Attrition • Resultingfromtoothtotoothcontact • Incisal and occlusalsurfaces • Physiologicmovement and contact of teethbytheage • Parafunctionalhabitsassuchasbruxism

  18. Attrition

  19. Abfraction

  20. Erosion • Extrinsic acids • Commonnly associated with acidic foods and drinks • Carbonated drinks • Citrus drinks • Citrus fruits • Wine • Intrinsic acids • Gastric reflux • Bulimia

  21. Erosion • Dietary habits • Prolonged contact

  22. Erosion

  23. Gingival recession • Incorrect tooth-brushing or flossing • Gingivitis • Periodontitis • Periodontal procedures • Gingival trauma (picking hard foods)

  24. Gingivalrecession

  25. Enamelfracture

  26. Toothwhitening • Post dentalbleachingsensitivityreported 55-75% of patients • Increasedtoothporiosity and removal of protective protein layeronthesurface of theteeth • Opened upporesinexposed dentin surfaces

  27. Developmentalanomaly • MolarIncisorHypomineralisation • Enamelhypoplasia • Dentin exposurebecause of weakenamel

  28. Differentialdiagnosis • Dentalcaries • Periodontaldesease • Dentalfractures • ReversibleorIrreversiblepulpitis • Traumaticocclusion • TMJ disorder

  29. Diagnosisindentinehypersensitivity • Tactilehypersensitivityscore: • YeapleProbe (forcemeasuredingrams (10-50g onexposuredsurface) • SchiffCold Air SensitivityScale, usedairblastsimulusonexposuredsurfacefor 1 sec. (scores: 0-3, no paintopainful) • Visual AnalogScale (VAS) subjectivepainintensitymeasurement (0-10, no paintoworstimaginablepain)

  30. Treatment principles • Desensitizing nerves • Occlusion of open tubules block the hydrodynamic mechanism

  31. Treatment I.Desensitizing the nerves • Potassium salts: • potassium nitrate 5% • potassium citrate 3,75% • Potassium chloride 5,5% • Depolarize the excited nervefibers, thus numbing the pain • compatible with Fluoride

  32. Treatment II.Occlusion of dentine tubules Strontium salts High Fluoride Stannosus fuoride HEMA / Glutaraldehyde Restorative resins Ca,Na Phosphosilicate Arginine és calcium carbonate Arginine és PVM/MA copolimer, és Pyrophosphates

  33. Special Tooth pastes

  34. Restorative resin desensitizers

  35. Fillingthelesions

  36. Thankyouforyourattention!

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