AKI (formerly ARF). 13–18% of all people admitted to hospital. Format for the Session. 8.10 Acute Kidney Injury (AKI) R ecognise AKI/potential for AKI Distinction from chronic renal failure Establish underlying pathophysiology (causes) Investigations Management P rognosis
Download Policy: Content on the Website is provided to you AS IS for your information and personal use and may not be sold / licensed / shared on other websites without getting consent from its author.While downloading, if for some reason you are not able to download a presentation, the publisher may have deleted the file from their server.
13–18% of all people admitted to hospital
8.10 Acute Kidney Injury (AKI)
Within the past 7 days:
50% of AKI
When there’s something bad, and it don’t look good
Who yagonna call?
Your SHO/reg and suggest a referral to Renal boffins for an opinion on haemodialysis
Acidosis (metabolic)Electrolyte changes (hyperkalemia)Ingested Toxins (barbiturates, salicylates, lithium, methanol)Overload (pulmonary oedema unresponsive to diuretics)Uremia symptoms (pericarditis, encephalopathy)
3.26 Abnormalities of sodium and potassium
By the end of Phase II students should be able to:
•initiate investigation of hypo and hyper natraemia and hypo and hyper kalaemia
•initiate management of these conditions