Hepatic Disorders: Hepatitis/Cirrhosis. Lisa Randall, RN, MSN, ACNS-BC RNSG 2432. Objectives. C ompare and contrast risk factors associated with hepatitis and cirrhosis Analyze the etiology and pathophysiology of hepatitis and cirrhosis
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Lisa Randall, RN, MSN, ACNS-BC
B Hepatic vein
C Hepatic artery
D Portal vein
E Common bile duct
G Cystic duct
*See Table 44.15
See table 44.17
Spontaneous bacterial peritonitis
bleeding, shock & infection
Later prevention of re-bleeding
sclerosant solution (ethanolamine oleate or sodium tetradecylsulphate)Complications
fever, dysphagia and chest pain, ulceration, stricture, and (rarely) perforation.
Fewer treatment sessions and complications than sclerotherapy.
Tube is inserted through the mouth
Correct placement within the stomach is checked by auscultation while injecting air through the gastric lumen
Gastric balloon is then inflated with 200 ml of air
Gastric balloon is pulled up against the esophagogastric junction, compressing the submucosalvarices
Tension is maintained by strapping a split tennis ball to the tube at the patient's mouth
gastric and esophageal ulceration
Sengstaken-Blakemore tube – has only 3 lumens