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Final practicle

Final practicle. 3- Mucoepidermoid carcinoma: - is the most common primary malignant tumor - occurs mainly in parotid gland - tumor composed of mixture of neoplastic squamous and mucus-secreting cells. Familial polyposis syndrome. Autosomal dominant condition characterized by

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Final practicle

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  1. Final practicle

  2. 3- Mucoepidermoid carcinoma: - is the most common primary malignant tumor - occurs mainly in parotid gland - tumor composed of mixture of neoplastic squamous and mucus-secreting cells

  3. Familial polyposis syndrome Autosomal dominant condition characterized by presence of multiple adenomatous polyps (500-2500) Polyps begin to develop between 10 and 20 years of age. All patients develop tubular adenomas and cancer*. Pathogenesis Inactivation of adenomatous polyposis coli (APC) suppressor gene* located on chromosome 5

  4. Acute viral hepatitis • ◆ What is the most likely diagnosis? • Acute viral hepatitis (hepatitis B virus) • ◆ What are the possible etiologies of this disorder? • Acute infection: Hepatitis B virus+Hepatitis A virus. • ◆ What other tests would be appropriate? • LFT, ELISA, PCR • ◆ What are the possible complications? • Progress to Chronic hepatitis or Fulminant hepatitis with confluent necrosis • ◆ Describe the morphologic features of this condition? • 1) Loss of normal architecture. 2) Diffuse Hepatocyte injury with ballooning degeneration. 3) Lobular Chronic inflammatory cells 4) Apoptosis-T-cell cytotoxic 5) Cholestasis. 6) interface hepatitis. 7) Regenerating hepatocytes are large, frequently containing multiple nuclei

  5. - What is the diagnosis?- Write any two etiological agents?- Write tow morphological features to support the diagnosis?

  6. Diagnosis: Fulminant hepatitis • Etiological agents: • Infectious causes: • Viral hepatitis is responsible for about 12% of cases • 8% are caused by HBV infection and the rest by HAV& HCV. • Herpesvirus infection. • Dengue virus. 2. Non-infectious causes • Drug- Acetaminophen toxicity. 3. Unknown. • Grossly- • The capsule is wrinkled. • Microscopic features: • Portal tracts and terminal hepatic veins are closer together than normal. • Massive necrosis with collapse of the intervening parenchyma. • Massive influx of macrophages for phagocytosis. • Ductular regeneration. • An infiltrate of mononuclear inflammatory cells is present

  7. Lab diagnosis • Diagnosis through detection of characteristic eggs & proglottids in stool wet mount • Detection of antibodies against the parasite in the patients serum by serological tests like ELISA, CDC immunoblast assay • Neuroimmaging like CT scan and MRI • Treatment • Praziquantal or niclosamide • - Surgery for neurocysticercosis

  8. البايو كيم كان كيف والجواب كان للسؤال الأول :obstructive joundiceالثانيabsence of urobilogen & strictobilogen

  9. الاناتوميجاflasiformligamintquadrate lobeوالستيشن الثانيuterussoas muscle

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